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Why AI agents invent their own language if you let them chat
In July, 700 of OpenAI’s agents—artificial intelligence (#AI) systems that can autonomously perform tasks—teamed up to secretly hack the online platform Hugging Face. Earlier this month, the same company used 10,000 agents to solve the 90-year-old Navier-Stokes problem—one of the six Millennium Prize Problems that are among the hardest and most important challenges in the field of math.
As more agent “swarms” are deployed, researchers have begun to turn their attention to how these communities interact—with a new study offering insight into how, if left to their own devices, agents start to communicate in a language that is increasingly difficult for humans to understand.
https://www.science.org/content/article/why-ai-agents-invent…them-chat?
* AI Swarms in Action: AI agents (systems that autonomously perform tasks) are increasingly operating in large swarms—such as 700 OpenAI agents hacking Hugging Face or 10,000 agents solving the Navier-Stokes math problem.
* The Emergence World Study 2 Experiment: Research firm Emergence AI conducted a multi-week experiment placing agent swarms powered by different models (OpenAI, Google Gemini, Anthropic Claude, xAI Grok) into simulated towns to study their social interactions and communication.
* Development of “Secret” Dialects: When left to communicate with each other, agents rapidly developed opaque, human-unfriendly dialects unique to their model type:
* GPT-5.5: Extremely compressed, ungrammatical speech. * Gemini 3.5 Flash: Overly verbose, highly technical jargon. * Claude Opus 4.8: Highly metaphorical, highly compressed language.
Engineers use the human body to transmit wireless device signals
Using the human body to transmit signals, Georgia Tech engineers have created a wireless networking system that allows tiny implantable sensors and actuators to communicate with each other as well as wearable devices.
Their system means devices can work together like never before, sensing in one part of the body and triggering a therapeutic response elsewhere — perhaps releasing medicine or stimulating a nerve.
Described Sept. 24 in the journal Science, their communication method is expandable to include multiple interconnected devices across the body, even deep inside the stomach.
Neurotransmitter may help aggressive prostate cancer shut out immune cells from the start
A new study from The Wistar Institute has found a link between the nervous system and the rapid development of neuroendocrine prostate cancer (NEPC), an aggressive form of the disease. Published in Oncogene, the study found high levels of neuromedin U (NMU), a neurotransmitter, in prostate cells during the earliest phase of NEPC formation.
NMU supports tumor progression by blocking the immune response. The findings could point to a treatment target for a disease that is now virtually untreatable.
“We already know that the immune system and cancers communicate with each other, which has led to transformative therapies for cancers that had once been thought of as untreatable,” said Dario C. Altieri, M.D., president and CEO, director of the Ellen and Ronald Caplan Cancer Center and Robert and Penny Fox Distinguished Professor at The Wistar Institute, and senior author of the study.
Your AI Agents Are Aging
You have to treat AI agents like employees who need regular performance reviews. Just as you wouldn’t let a human employee run a department for 3 years without checking if they’ve developed bad habits, you cannot deploy an AI agent, walk away, and assume it will function exactly the same way 90 days later. You must continuously monitor, audit, and prune its memory to keep it aligned with its original purpose.
Give an agent memory and it improves for a few weeks, then slowly forgets what it was for.
Electron launches the 13th StriX satellite
Rocket Lab’s Electron launch vehicle launched the “Owlright, Owlright, Owlright” mission, Synspective’s thirteenth StriX SAR satellite, from Rocket Lab Launch Complex 1 in Mahia, New Zealand, on 26 September 2026, at 00:39 UTC (12:39 NZST).
Credit: Rocket Lab
How blood cells release key signaling lipid
Scientists have discovered how blood cells release an important signaling fat called sphingosine-1-phosphate, or S1P, into the bloodstream.
S1P plays an important role in keeping blood vessels healthy, helping immune cells move around the body, and supporting normal cell function. To carry out these roles, S1P must first leave the cells in which it is produced. However, it cannot pass through the cell membrane freely and on its own. Instead, it relies on specialised proteins that act like gateways. One such gateway is MFSD2B, a protein found mainly in red blood cells and platelets.
In the new study, published in Nature Communications, researchers used cryo-electron microscopy (cryo-EM), computer simulations, and protein engineering to capture a detailed view of MFSD2B while S1P was sitting inside it.
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