Boyer, Blaye et al. show that CD52-expressing immunosuppressive myeloid cells can shape breast tumor plasticity beyond immune evasion. They show that this myeloid subset delivers membrane-bound TGF-β1 via cell contact to generate mesenchymal-like cancer stem cells in luminal breast cancer, and TGF-β blockade prevents this stemness program.
What if the Borg discovered a way to travel beyond the boundaries of their universe?
Not faster warp.
Not another transwarp conduit.
But a doorway into infinite realities.
In this video, we explore one of the most terrifying possibilities in Star Trek: Why did the Borg never conquer the multiverse?
With their transwarp technology, adaptive intelligence, and endless hunger for perfection, the Borg seem uniquely positioned to expand beyond a single universe. Every reality could contain new civilizations to assimilate, unimaginable technologies to steal, and knowledge beyond anything the Collective has ever encountered.
Seven years after the original 2019 report, the reported 3.6 liters per hour per square meter remains an interesting benchmark for understanding the potential of solar purification research. The lasting value of the work is not simply the numerical comparison with commercial solar stills but the broader principle behind it: materials can be engineered to make renewable-energy technologies more productive.
Some of nature’s most important chemical reactions rely on the coupled movement of negatively and positively charged particles. These processes play central roles in photosynthesis, catalysis and biological energy conversion yet remain difficult to observe.
Now, a research team led by the Department of Energy’s Pacific Northwest National Laboratory, in collaboration with colleagues at SLAC National Accelerator Laboratory and several academic labs, has captured snapshots of these events triggered when light strikes a molecule.
The findings, published in Nature Communications, could help researchers better understand and ultimately design better flow batteries, fuel cells and catalysts.
In a tale of identities as divergent as “The Prince and the Pauper,” a well-known cancer-causing gene also influences cells in a zombie-like state meant to prevent cancer.
Scientists at Sanford Burnham Prebys Medical Discovery Institute and an international team of collaborators published findings Aug. 20, 2026, in Nature Aging that unpack this biological paradox, showing that this cell proliferation gene played a distinct role in cells that no longer proliferate. It served as a driver of chronic inflammation linked to age-related disease, making it a promising target for future therapies to reduce sustained inflammation and promote healthier aging.
The code for producing the protein cyclin D1 is carried by the gene CCND1. This gene made waves in the field of oncology in the early-to mid-1990s when overexpressing it was shown to cause cancer. Normally, cyclin D1 governs the activity of signaling molecules needed to push cells along the cell cycle toward replicating their DNA and dividing to make new cells. This process goes haywire and becomes hyperactive in cancer, but it is nonexistent in zombie-like senescent cells.
From the starting gun to the final whistle, time is central to our lives. We shape our days with diaries and timetables, even if the trains do not always take note. Yet the nature of time itself is elusive and unknown. A new year begins, and we get older, but however closely we look we cannot observe time, only its passing. Time is equally puzzling to science. Central to its laws from Newton to Einstein, science is unable to describe time itself. Strangely, in the so-called