Lifeboat Foundation: Safeguarding Humanity
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Category: life extension – Page 6

We are currently facing the possibility of achieving immortality for humans by 2030. This prediction comes from renowned futurist Ray Kurzweil, who has a history of making accurate predictions. He anticipates that with the ongoing progress in genetics, robotics, and nanotechnology, we will soon have nanobots coursing through our bloodstream, which could enable us to live forever. It’s truly remarkable to consider that this could be a reality within just seven years.

Nanobots, which are small robots sized between 50–100 nm in width, are currently being used in various clinical medical applications. They are used in research as DNA probes, imaging materials for cells, and targeted delivery vehicles for cells. According to Kurzweil, nanobots represent the future of medicine.

They will be capable of repairing our bodies at a cellular level, making us resistant to diseases, aging, and, ultimately death. Additionally, he theorizes that humans may be able to transfer their consciousness into digital form, leading to immortality.

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Continue reading “Supplements, Diet That Correspond To A 17y Younger Biological Age (Test #3 in 2025)” | >

A new study suggests that the questions you ask and your interest in lifelong learning may help protect against Alzheimer’s disease. What’s the secret to aging well? It might be as simple as staying curious. A new international study, including researchers from UCLA, found that certain types o

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Manipulating senescent cells by eliminating them or by modifying their activity has attracted huge interest for its potential to delay or even treat many age-related diseases, and to improve healthy aging. Mitochondria, and in particular their calcium levels, have emerged as key regulators of cellular senescence, cell death and the balance between the two, and might constitute targets for novel strategies to stifle the viability or properties of senescent cells.

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Scientists in Europe have tested an anti-aging drug cocktail in mice and found that it extended the animals’ lifespans by around 30 percent. The mice stayed healthier for longer too, with less chronic inflammation and delayed cancer onset.

The two drugs are rapamycin and trametinib, which are both used to treat different types of cancer. Rapamycin is also often used to prevent organ rejection, and has shown promise in extending lifespans in animal tests. Trametinib, meanwhile, has been shown to extend the lifespan of fruit flies, but whether that worked in larger animals remained to be seen.

So for a new study, a research team led by scientists from the Max Planck Institute in Germany investigated how both drugs, on their own and together, could extend lifespan in mice.

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As the population ages, multimorbidity, or when a patient has multiple diseases at once, is becoming increasingly common. The onset of one disease increases the risk of developing other diseases, making it necessary to investigate how a range of risk factors together affect such accumulation. Prior studies have focused on individual risk factors and related individual diseases.

A study explored how the risk factors measured from birth to middle age and unmeasured, or latent, factors covering the entire lifespan predict and explain the incidence of chronic diseases in eight organ systems from middle to old age: the cardiovascular, metabolic, gastrointestinal, musculoskeletal, respiratory, neurological and psychiatric systems, and the sensory organs.

The study, published in The Lancet Healthy Longevity journal, analyzed 22 risk factors, including age, sex, (e.g., size at birth, early childhood growth, childhood wartime evacuee status), socioeconomic factors (e.g., socioeconomic status in childhood, income in adulthood), lifestyle factors (e.g., smoking, , , diet), clinical measurements and biomarkers (e.g., body mass index, , blood glucose).

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The process of necrosis, a form of cell death, may represent one of the most promising ways to change the course of human aging, disease and even space travel, according to a new study by researchers at UCL, drug discovery company LinkGevity and the European Space Agency (ESA).

In the study, published in Oncogene, an international team of scientists and clinicians explore the potential of —when cells die unexpectedly as a result of infection, injury or disease—to reshape our understanding and treatment of age-related conditions.

Challenging prevailing views, the paper brings together evidence from cancer biology, , kidney disease, and space health to argue that necrosis is not merely an endpoint, but a key driver of aging that presents an opportunity for intervention.

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