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New research reveals that spinning quasiparticles, or magnons, light up when paired with a light-emitting quasiparticle, or exciton, with potential quantum information applications.

All magnets contain spinning quasiparticles called magnons. This is true of all magnets from the simple souvenirs hanging on your refrigerator to the discs that give your computer memory storage to the powerful versions used in research labs. The direction one magnon spins can influence that of its neighbor, which in turn affects the spin of its neighbor, and so on, yielding what are known as spin waves. Spin waves can potentially transmit information more efficiently than electricity, and magnons can serve as “quantum interconnects” that “glue” quantum bits together into powerful computers.

Although magnons have enormous potential, they are often difficult to detect without bulky pieces of lab equipment. According to Columbia researcher Xiaoyang Zhu, such setups are fine for conducting experiments, but not for developing devices, such as magnonic devices and so-called spintronics. However, seeing magnons can be made much simpler with the right material: a magnetic semiconductor called chromium sulfide bromide (CrSBr) that can be peeled into atom.

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Two big players in computing and research are trying to lay the groundwork for a future quantum internet.

Amazon Web Services (AWS) is teaming up with Harvard University to test and develop strategies for networking together quantum technologies. Their partnership was announced today, and is a continuation of AWS’ goals to create a communications channel between the quantum computers that it is also working on in parallel.

During the three-year research alliance, funding from Amazon will support research projects at Harvard that focus on quantum memory, integrated photonics, and quantum materials, and help upgrade infrastructure in Harvard’s Center for Nanoscale Systems.

You need to wait till 2023 to get them though.

Lenovo has unveiled its T1 Glasses at its Tech Life 2022 event and promises to place a full HD video-watching experience right inside your pockets, a company press release stated.

Mobile computing devices have exploded in the past few years as gaming has become more intense, and various video streaming platforms have gathered steam. The computing power of smartphones and tablets has increased manifold. Whether you want to ambush other people in an online shooting game or sit back and watch a documentary in high-definition, a device in your pocket can help you do that with ease.

Circa 2019 face_with_colon_three


By Tyler Benster.

Neuroscientists have a dizzying array of methods to listen in on hundreds or even thousands of neurons in the brain and have even developed tools to manipulate the activity of individual cells. Will this unprecedented access to the brain allow us to finally crack the mystery of how it works? In 2017, Jonas and Kording published a controversial research article, “Could a Neuroscientist Understand a Microprocessor?” that argues maybe not. To make their point, the authors turn to their “model organism” of choice: a MOS 6502 processor as popularized by the Apple I, Commodore 64, and Atari Video Game System. Jonas and Kording argue that for an electrical engineer, a satisfying description of the processor would break it into modules, like an adder or subtractor, and submodules, like the transistor, to form a hierarchy of information processing. They suggest that, while popular methods from neuroscience might reveal interesting structure in the activity of the brain, researchers often use techniques that would fail to reveal a hierarchy of information processing if applied to the (presumably much simpler) computer processor.

For example, neuroscientists have long used lesions, or turning off or destroying a part of the brain, to try to find links between that brain region and particular behaviors. In one particularly striking experiment, the authors mimicked this classic technique by simulating the processor as it performed one of four “behaviors”: Donkey Kong, Space Invaders, Pitfall, and Asteroids. They then systematically removed one transistor, and reported which (if any) of the behaviors could still be performed (i.e. did the game boot?) The elimination of 1,565 transistors have no impact, while 1,560 inhibit all behaviors, and indeed a subset of transistors make only one game impossible. Perhaps these are the Donkey Kong transistors, the authors coyly suggest, before concluding that the “causal relationship” is highly superficial.

A new device has been fabricated that can demonstrate the quantum anomalous Hall effect, in which tiny, discrete voltage steps are generated by an external magnetic field. This work may enable extremely low-power electronics, as well as future quantum computers.

If you take an ordinary wire with running through it, you can create a new electrical voltage perpendicular to the flow of current by applying an . This so-called Hall effect has been used as part of a simple magnetic sensor, but the sensitivity can be low.

There is a corresponding quantum version, called the quantum anomalous Hall effect that comes in defined increments, or quanta. This has raised the possibility of using the quantum anomalous Hall effect for the purpose of constructing new highly conductive wires or even quantum computers. However, the physics that leads to this phenomenon is still not completely understood.

NASA — National Aeronautics and Space Administration has tapped SiFive and Microchip Technology Inc. to create a space-centric RISC-V processor: the High-Performance Spaceflight Computing chip. At heart of the HPSC will be SiFive’s X280 64-bit RISC-V cores, which include ML acceleration capabilities.


Designed to replace existing systems still using a processor design from 1997, the RISC-V-powered chip will offer 100 times the performance.

O.o!!!


According to recent research, the protein CHIP can control the insulin receptor more effectively while acting alone than when in a paired state. In cellular stress situations, CHIP often appears as a homodimer – an association of two identical proteins – and mainly functions to destroy misfolded and defective proteins. CHIP thus cleanses the cell. In order to do this, CHIP works with helper proteins to bind a chain of the small protein ubiquitin to misfolded proteins.

As a result, the cell detects and gets rid of defective proteins. Furthermore, CHIP controls insulin receptor signal transduction. CHIP binds to the receptor and degrades it, preventing the activation of life-extending gene products.

Researchers from the University of Cologne have now shown via tests using human cells and the nematode Caenorhabditis elegans that CHIP can also label itself with ubiquitin, preventing the formation of its dimer. The CHIP monomer regulates insulin signaling more effectively than the CHIP dimer. The research was conducted by the University of Cologne’s Cluster of Excellence for Cellular Stress Responses in Aging-Associated Diseases (CECAD) and was recently published in the journal Molecular Cell.