Dietary intakes of flavonols, flavones and isoflavones by Japanese women and the inverse correlation between quercetin intake and plasma LDL cholesterol concentration. https://pubmed.ncbi.nlm.nih.gov/10958819/
NAD and the aging process: Role in life, death and everything in between.
A bitter divorce battle in Russia has led to cryogenically frozen bodies being seized in an extraordinary day-time raid.
Staff of a company set up by Valeria Udalova, 59 grabbed the remains of people — including from Britain and the US — who paid thousands of pounds hoping to be brought back to life when science advances to allow this.
She and her team raided the company run by her ex-husband Danila Medvedev, 41 near Moscow, which is home to Russia’s leading cryo-storage facility, say reports.
#ElonMusk is on a better path than Bezos is partly because he’s working on his brain chip and once that brain chip has been made dubbed complete he can make it so it works with tech and we can merge Tech with people which means that we can make a life out of tech and have our body be completely made robotic and as long as we have materials like synthetic blood that feeds and sustains the brain with the proper nutrients to stay alive. Theoretically u could live much longer. He seems to have the robot body and robot made and chip almost complete now he needs the synthetic sustainable blood for a human brain 🧠 implant into the robot tech body using Brian chip to control it. Immortality In theory the brain doesn’t have to be in the robotic body it can still be connected to the body through a central location via #StarLink #Robot links https://fb.watch/7UAKDX92Vh/https://fb.watch/7Uyo7JYdok/ “Life is a video game in that aspect” But even without the brain merge you will be able to pair our minds with these AI robots and use them on 🌎 earth or beyond like a ship to #Mars or any other place that can get the signal from the brain to the robotic body.
What might happen after the Cybernetic Singularity? Can we refine our theological philosophies in light of new evidence? Are we alone in the Universe? Can you achieve Cybernetic Immortality? When and how can we transcend the human condition? These are some of the questions addressed in my new book THEOGENESIS: Transdimensional Propagation & Universal Expansion. This awe-inspiring volume is to be released on October 1 2021 as part of The Cybernetic Theory of Mind series and is available now to pre-order on Amazon. #THEOGENESIS #TransdimensionalPropagation #UniversalExpansion #CyberneticTheoryofMind #cybernetics #theology #futurism #posthumanism
Ecstadelic Media Group announces the release of the next book in The Cybernetic Theory of Mind series by Alex M. Vikoulov ― THEOGENESIS: Transdimensional Propagation & Universal Expansion ― available as a Kindle eBook on Amazon.
In this video Dr. Katcher reveals his thought on the future of aging if E5 is fulfils on its promise.
Dr Katcher’s book is on Amazon. The Illusion of Knowledge: The paradigm shift in aging research that shows the way to human rejuvenation. https://amzn.to/3jJ5deD
Dr Harold Katcher is one of the discovers of the human breast cancer gene BRCA1, and has thousands of citations in the scientific literature with publications ranging from protein structure to bacteriology, bioinformatics and biochemistry. He was the Academic Director for Natural Sciences of the University of Maryland Global Campus and is now the Chief Scientific Officer at Yuvan Research Inc, a company working on the development of rejuvenation treatments.
Dr Katcher’s new book, the Illusion of Knowledge, the paradigm shift in aging research that shows the way to human rejuvenation will be launched on 4th September 2021 and is already available in electronic form. The book launch will take place at The Book Passage in the Ferry Building in San Francisco at 3:00 pm Pacific Time.
************************************************************ Health claims Disclosure: Information provided on this video is not a substitute for direct, individual medical treatment or advice. Please consult with your doctor first. Products or services mentioned in this video are not a recommendation.
Summary: A new study on aging reveals a surprising discovery about the connection between protein shape and mitochondrial health.
Source: Buck Institute.
Every cell in the body goes through thousands of chemical reactions each day, and each reaction involves tiny protein molecules folded into precise shapes to perform their functions. Misfolded proteins underlie some of the most common and devastating diseases of aging, like Alzheimer’s and Parkinson’s. A major focus of aging research is discovering ways to maintain protein shape and prevent misfolded proteins from wreaking havoc on cellular function.
Dr. Ezcurra received her PhD from the Karolinska Institute in 2011. Her PhD research was a collaborative project between Karolinska and the Medical Research Council Laboratory of Molecular Biology at Cambridge, where she studied neural circuits and behavior using C. elegans in the lab of Dr. Bill Schafer.
During her PhD, Dr. Ezcurra identified extra-synaptic mechanisms by which nutritional status modulates nociception, involving neuro-peptidergic and dopaminergic signaling. She went on to do a postdoc working on aging with Dr. David Gems at University College London.
During her postdoc, Dr. Ezcurra developed methods to monitor the development of multiple age-related diseases in-vivo in C. elegans, leading to the discovery of a previously unknown process, Intestinal Biomass Conversion. This mechanism enables the C. elegans intestine to be broken down to produce vast amounts of yolk, resulting in poly-morbidity and mortality in aging nematodes. This work illustrates how aging and age-related diseases can be the result of run-on of wild-type gene function, rather than stochastic molecular damage.
People with rare disorders that cause shortened telomeres—protective caps that sit at the end of chromosomes—may be more likely to have blood cancers such as leukemia or myelodyplastic syndrome. Now, Johns Hopkins Medicine scientists have discovered several “self-correcting” genetic mutations in bone marrow that may protect such patients from these cancers.
In a study published online August 3 2021, in the Journal of Clinical Investigation, the researchers also suggest these mutations can serve as biomarkers that may indicate if patients with short telomere syndromes are likely to develop blood cancers.
“These are the most common cancers we see in patients with short telomere syndromes,” says Mary Armanios, M.D., director of the Telomere Center and professor of oncology at the Sidney Kimmel Comprehensive Cancer Center at Johns Hopkins. “We know that at a certain point, the cells of patients with shortened telomeres either become cancerous or stay healthy.”
Summary: Researchers have identified 2,000 genes in humans linked to longevity. The genes are associated with biological mechanisms that drive the prolongation of life in mammals, including DNA repair, coagulation, and immune response.
Source: UPF Barcelona.
What determines the life expectancy of each species? This is a fundamental and highly complex question that has intrigued the field of research throughout history. From the evolutionary point of view, the major cause of these differences between species lies in their ecological adaptations. For example, life expectancy is longer in species adapted to living in trees, underground, or with large body mass, since all these adaptations reduce mortality by predation.
Youthereum talking about rejuvenation, funding, there is a history lesson here but the modern look starts at 38:01.
My overview of the history of partial reprogramming — a novel approach to epigenetic rejuvenation that uses short bursts of Yamanaka factors expression to periodically roll back the epigenetic state of cells to a younger pattern.
