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At-Home Metabolomics: https://www.iollo.com?ref=michael-lustgarten.
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Epigenetic, Telomere Testing: https://trudiagnostic.com/?irclickid=U-s3Ii2r7xyIU-LSYLyQdQ6…M0&irgwc=1
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NAD+ Quantification: https://www.jinfiniti.com/intracellular-nad-test/

(/ ˈ m ʌr i ˈ ɡ ɛ l ˈ m æ n / ; September 15, 1929 – May 24, 2019) [ 3 ] [ 4 ] [ 5 ] [ 6 ] was an American theoretical physicist who played a preeminent role in the development of the theory of elementary particles. Gell-Mann introduced the concept of quarks as the fundamental building blocks of the strongly interacting particles, and the renormalization group as a foundational element of quantum field theory and statistical mechanics. He played key roles in developing the concept of chirality in the theory of the weak interactions and spontaneous chiral symmetry breaking in the strong interactions, which controls the physics of the light mesons. In the 1970s he was a co-inventor of quantum chromodynamics (QCD) which explains the confinement of quarks in mesons and baryons and forms a large part of the Standard Model of elementary particles and forces.

Murray Gell-Mann received the 1969 Nobel Prize in Physics for his work on the theory of elementary particles.

Gestational diabetes is a disorder characterized by abnormally high levels of blood glucose (also called blood sugar) during pregnancy.

Affected women do not have diabetes before they are pregnant, and most of these women go back to being nondiabetic soon after the baby is born. Gestational diabetes is often discovered during the second trimester of pregnancy.

The disease has a 30 to 70 percent chance of recurring in subsequent pregnancies.

Gestational diabetes mellitus (GDM) is influenced by both genetic and environmental factors. Polymorphisms in genes related to glucose metabolism and insulin signaling, such as TCF7L2, have been associated with increased risk. This gene influences insulin secretion and glucose production, affecting the body’s ability to regulate blood sugar during pregnancy. Other implicated genes include GCK, encoding glucokinase, and MTNR1B, involved in melatonin receptor signaling.

More information on genetic factors that contribute to the development of GDM is provided in the link below.