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Critical vm2 sandbox bug lets attackers execute code on hosts

A critical vulnerability in the popular Node.js sandboxing library vm2 allows escaping the sandbox and executing arbitrary code on the host system.

The security issue is tracked as CVE-2026–26956 and has been confirmed to impact vm2 version 3.10.4, although earlier releases may also be vulnerable. Proof-of-concept (PoC) exploit code has been published.

In the security advisory, the maintainer says that the issue only impacts environments with Node.js 25 (confirmed on Node.js 25.6.1) that have enabled WebAssembly exception handling and JSTag support.

Hackers abuse Google ads for GoDaddy ManageWP login phishing

A phishing campaign delivered through Google sponsored search results is targeting credentials for ManageWP, GoDaddy’s platform for managing fleets of WordPress websites.

The threat actor is using an adversary-in-the-middle (AitM) approach where the fake login page acts as a real-time proxy between the victim and the legitimate ManageWP service.

ManageWP is a centralized remote administration platform for WordPress websites, enabling users to manage multiple sites from a single panel instead of logging into separate dashboards. Common users include web developers, web agencies managing client sites, and enterprises.

New Cisco DoS flaw requires manual reboot to revive devices

The company advised customers at the time to contact its Technical Assistance Center (TAC) to have them brought back online, as this required manual intervention.

Last year, Cisco patched another DoS vulnerability (CVE-2025–20115) that allowed attackers to crash the Border Gateway Protocol (BGP) process on IOS XR routers with a single BGP update message.

DAEMON Tools devs confirm breach, release malware-free version

Disc Soft Limited, the maker of DAEMON Tools Lite, confirmed that the software had been trojanized in a supply chain attack and released a new, malware-free version.

“Within less than 12 hours of identifying the issue, we were able to implement a solution. Based on our current findings, the issue was limited to the free DAEMON Tools Lite version and did not affect any of our other products,” Disc Soft told BleepingComputer.

“We have not identified evidence supporting claims that all DAEMON Tools users were impacted, and at this stage, we are not in a position to confirm any impact on paid versions customers. Our current analysis indicates that DAEMON Tools Pro and DAEMON Tools Ultra were not affected and absolutely safe.”

Study: Gene circuits reshape DNA folding and affect how genes are expressed

When a gene is turned on in a cell, it creates a ripple effect along the DNA strand, changing the physical structure of the strand. A new study by MIT researchers shows that these ripples can stimulate or suppress neighboring genes.

These effects, which result from the winding or unwinding of neighboring DNA, are determined by the order of genes along a strand of DNA. Genes upstream of the active gene are usually turned up, while those downstream are inhibited.

The new findings offer guidance that could make it easier to control the output of synthetic gene circuits. By altering the relative ordering and arrangement of genes, or “gene syntax,” researchers could create circuits that synergize to maximize their output, or that alternate the output of two different genes.

Zuckerberg Trying to Simulate Human Biology at the Cellular Level

Mark Zuckerberg is following a path paved by fellow billionaires Bill Gates and Warren Buffet: laundering his untold billions through a health research prestige project.

Called the Chan Zuckerberg Biohub — his wife Priscilla Chan, a pediatrician, is also involved — the foundation’s stated long-term mission is to “cure and prevent all disease through AI-powered biology, frontier research, and state-of-the-art technology.”

True to those enormous goals, the Biohub recently announced a $500 million investment into AI models of human cells, specifically, in order to “accelerate the cure and prevention of all diseases,” Euronews reported.

ALS and Huntington Disease: Unraveling the Connections between TDP-43 and Huntingtin

Amyotrophic lateral sclerosis (ALS) and Huntington disease (HD) are lethal neurodegenerative diseases affecting motor function. Though their etiology and pathology are distinct, recent evidence suggests commonalities between TAR DNA-binding protein (TDP-43), which is associated with 97% of ALS cases, and huntingtin (HTT), the causative protein of HD. ALS is a heterogeneous, lethal neurodegenerative disease characterized by the progressive loss of upper and lower motor neurons, as well as brainstem and spinal cord degeneration. The causes of ALS are complex, variable, and, in some cases, unknown, but most cases involve mislocalization of the protein TDP-43. In contrast, HD is a monogenic, autosomal dominant, lethal neurodegenerative disease caused by polyglutamine expansion in HTT protein and characterized by the progressive loss of neurons in the brain, particularly in the striatum, which results in motor, cognitive, and behavioral changes. Although HD is not typically associated with motor neuron loss, recent evidence suggests a link between HTT and TDP-43 within the context of both ALS and HD, as well as links to related neurodegenerative diseases, such as frontotemporal dementia (FTD) and spinocerebellar ataxia type 2 (SCA2). Herein, we discuss confirmed cases of concurrent ALS and HD and the overlap of underlying disease mechanisms that potentially contribute to the onset and progression of these two devastating neurodegenerative diseases, with a focus on commonalities between TDP-43 and HTT. We propose that elucidating these commonalities will aid in the identification of broad-spectrum disease risk factors and potential overlapping treatment targets.

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