Penny for your thoughts.
Backed by $54 million, Kernel aims to turn an esoteric science into big business.
Category: neuroscience – Page 711
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Elucidating how the brain controls peripheral organs in the fight against infection is crucial for our understanding of brain–body interactions. A study in mice reveals one such pathway worthy of further investigation. Neurons in stress-responsive brain regions boost plasma-cell formation.
A study just released by Columbia University Mailman School of Public Health is reporting a blood-DNA-methylation measure that is sensitive to variation in the pace of biological aging among individuals born the same year. The tool—DunedinPoAm—offers a unique measurement for intervention trials and natural experiment studies investigating how the rate of aging may be changed by behavioral or drug therapy, or by changes to the environment. The study findings are published online in the journal eLife.
“The goal of our study was to distill a measurement of the rate of biological aging based on 12-years of follow-up on 18 different clinical tests into a blood test that can be administered at a single time point.” said lead author Daniel Belsky, Ph.D., assistant professor of epidemiology at Columbia Mailman School and a researcher at the Columbia Aging Center.
Midlife adults measured to be aging faster according to the new measurement showed faster declines in physical and cognitive functioning and looked older in facial photographs. Older adults measured to be aging faster by the tool were at increased risk for chronic disease and mortality. In other analyses, the researchers showed that DunedinPoAm captured new information not measured by proposed measures of biological aging known as epigenetic clocks, that 18-year-olds with histories of childhood poverty and victimization showed faster aging as measured by DunedinPoAm, and that DunedinPoAm predictions were disrupted by a caloric restriction intervention in a randomized trial.
World-first research led by neuroimaging expert Dr. Jiyang Jiang at UNSW’s Centre for Healthy Brain Ageing (CHeBA) has found that those aged 95 and over demonstrated more activation between the left and ride side of their brain than their younger counterparts.
Given the prevalence of dementia increases with age, near-centenarians and centenarians without dementia are generally considered as models of successful aging and resistance against age-related cognitive decline.
“We wanted to see if there was something particularly special about the brain’s functional connectivity of those aged 95 and older that helps them preserve brain function into the 11th decade of their life,” says Dr. Jiang.
Fyodor Rouge, this is the church I was telling you about.
The Bradenton-based organization falsely claims that the treatment is effective for a number of conditions, including Alzheimer’s disease, brain disease, cancer, HIV and AIDS, according to the Food and Drug Administration.
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Electronic devices which mimic the functionality of biological synapses are a large step to replicate the human brain for neuromorphic computing and for numerous medical research investigations. One of the representative synaptic behaviors is paired-pulse facilitation (PPF). It has been widely investigated because it is regarded to be related to biological memory. However, plasticity behavior is only part of the human brain memory behavior.
Here, we present a phenomenon which is opposite to PPF, i.e., paired-pulse inhibition (PPI), in nano oxide devices for the first time. The research here suggests that rather than being enhanced, the phenomena of memory loss would also be possessed by such electronic devices. The device physics mechanism behind memory loss behavior was investigated. This mechanism is sustained by historical memory and degradation manufactured by device trauma to regulate characteristically stimulated origins of artificial transmission behaviors.
Under the trauma of a memory device, both the signal amplitude and signal time stimulated by a pulse are lower than the first signal stimulated by a previous pulse in the PPF, representing a new scenario in the struggle for memory. In this way, more typical human brain behaviors could be simulated, including the effect of age on latency and error generation, cerebellar infarct, trauma and memory loss pharmacological actions (such as those caused by hyoscines and nitrazepam).
Rats and equations help researchers develop a theory of how our brains keep track of when events took place.
Scientists have discovered that eyes and brains in rodents seem to have uncannily similar drainage systems used for self cleaning, and there’s reason to think this might apply to us, too.
This sort of maintenance is necessary to wash away waste cells and fluids, and we know that brains make use of a tiny network of pipes known as the glymphatic system, similar to the lymphatic system that clears out rubbish from the rest of the body.
New tests on mice and rats show that the structures at the back of their eyes — like the optic nerve and the retina — take a page or two from the glymphatic system playbook. In the absence of the standard lymphatic vessels, they funnel waste products through a network a lot like the one the brain uses.
Help NYC artist Maria Alekseev
Posted in biotech/medical, employment, finance, food, genetics, health, neuroscience
Maria became the very first COVID-19 patient to use Stem Cell Neurotherapy for COVID-19. In about 5 days, she will began to feel the healing effects of generating new lung cells which will eliminate her breathing problems.
We repurposed some tools from the Stem Cell Therapy for Cancer/Brain Tumor. Those tools are T-Cells, B-Cells, and Natural Killer Cells. Instead of programming those cancer killing cells to attack cancer cells, we have programmed them to seek out, identify, attack, and destroy all the Coronavirus cells in the entire body.
Stem Cell Neurotherapy sends therapeutic messages, e.g., “your stem cells are transforming into new cells for the lungs, liver, and kidneys” to the DNA inside the nucleus of stem cells. Inside the nucleus, the DNA receives the message and transmits it to the RNA, which translates the message into genetic code.
The genes inside the stem cells transmit the coded message to the proteins, which are converted by the mitochondria into ATP, which provides the energy for the coded message to transform the stem cells into a new set of lung cells, as well as new cells for the kidneys and liver.
These new cells in the lungs, kidneys, and liver will replace the cells that were infected by the COVID-19 virus. This results in the elimination of the coughing, fever, headaches, diarrhea, and breathing problems.
Final edition of stem cell neurotherapy for COVID-19.
We have just finished the Final Edition of Stem Cell Neurotherapy for COVID-19. We repurposed some tools from the Stem Cell Therapy for Cancer/Brain Tumor. Those tools are T-Cells, B-Cells, and Natural Killer Cells. Instead of programming those cancer killing cells to attack cancer cells, we have programmed them to seek out, identify, attack, and destroy all the Coronavirus cells in the entire body.
We still program the stem cells to transform themselves into new cells for the lungs, liver, and kidneys to replace those cells infected by the Coronavirus.
So, stem cells have a dual function in this therapy:
A. Destruction of the Coronavirus cells