Lifeboat Foundation

A team of scientists, led by Natural History Museum postdoctoral researcher Dr. Anne-Claire Fabre, have conducted the first study on how metamorphosis has influenced the evolution of salamanders.

Using micro-CT scanning to study the skulls of this group of animals, the team were able to build a huge dataset of 148 species of salamanders and used cutting-edge methods to describe the shape of the skull with nearly 1000 reference points, known as landmarks.

Dr. Fabre said, “Most studies of this kind are limited to just a few dozen landmarks. Our study is the first large-scale investigation of this incredibly diverse group. We have captured the shape of the skull in such great detail that it has allowed us to learn more than ever before about how these creatures evolved.”

Scientists identify an important protein that increases “bacterial virulence,” when mutated, changing harmless bacteria to harmful ones.

As far as humans are concerned, bacteria can be classified as either harmful, pathogenic bacteria and harmless or beneficial non-pathogenic bacteria. To develop better treatments for diseases caused by pathogenic bacteria, we need to have a good grasp on the mechanisms that cause some bacteria to be virulent. Scientists have identified genes that cause virulence, or capability to cause disease, but they do not fully know how bacteria evolve to become pathogenic.

To find out, Professor Chikara Kaito and his team of scientists from Okayama University, Japan, used a process called experimental evolution to identify molecular mechanisms that cells develop to gain useful traits, and published their findings in PLoS Pathogens. “We’re excited by this research because no one has ever looked at virulence evolution of bacteria in an animal; studies before us looked at the evolution in cells,” said Prof Kaito.

Every year, 2 million black hole mergers are missed — Australian scientists work out how to detect them, revealing a lost 8 billion light-years of Universe evolution.

Last year, the Advanced LIGO –VIRGO gravitational-wave detector network recorded data from 35 merging black holes and neutron stars. A great result — but what did they miss? According to Dr. Rory Smith from the ARC Centre of Excellence in Gravitational Wave Discovery at Monash University in Australia — it’s likely there are another 2 million gravitational wave events from merging black holes, “a pair of merging black holes every 200 seconds and a pair of merging neutron stars every 15 seconds” that scientists are not picking up.

Dr. Smith and his colleagues, also at Monash University, have developed a method to detect the presence of these weak or “background” events that to date have gone unnoticed, without having to detect each one individually. The method — which is currently being test driven by the LIGO community — “means that we may be able to look more than 8 billion light-years further than we are currently observing,” Dr. Smith said.

The expansion of the human brain during evolution, specifically of the neocortex, is linked to cognitive abilities such as reasoning and language. A certain gene called ARHGAP11B that is only found in humans triggers brain stem cells to form more stem cells, a prerequisite for a bigger brain. Past studies have shown that ARHGAP11B, when expressed in mice and ferrets to unphysiologically high levels, causes an expanded neocortex, but its relevance for primate evolution has been unclear.

Researchers at the Max Planck Institute of Molecular Cell Biology and Genetics (MPI-CBG) in Dresden, together with colleagues at the Central Institute for Experimental Animals (CIEA) in Kawasaki and the Keio University in Tokyo, both located in Japan, now show that this human-specific gene, when expressed to physiological levels, causes an enlarged neocortex in the common marmoset, a New World monkey. This suggests that the ARHGAP11B gene may have caused neocortex expansion during human evolution. The researchers published their findings in the journal Science.

The human neocortex, the evolutionarily youngest part of the cerebral cortex, is about three times bigger than that of the closest human relatives, chimpanzees, and its folding into wrinkles increased during evolution to fit inside the restricted space of the skull. A key question for scientists is how the human neocortex became so big. In a 2015 study, the research group of Wieland Huttner, a founding director of the MPI-CBG, found that under the influence of the human-specific gene ARHGAP11B, mouse embryos produced many more neural progenitor cells and could even undergo folding of their normally unfolded neocortex. The results suggested that the gene ARHGAP11B plays a key role in the evolutionary expansion of the human neocortex.

Last year, the Advanced LIGO-VIRGO gravitational-wave detector network recorded data from 35 merging black holes and neutron stars. A great result—but what did they miss? According to Dr. Rory Smith from the ARC Centre of Excellence in Gravitational Wave Discovery at Monash University in Australia—it’s likely there are another 2 million gravitational wave events from merging black holes, “a pair of merging black holes every 200 seconds and a pair of merging neutron stars every 15 seconds” that scientists are not picking up.

Dr. Smith and his colleagues, also at Monash University, have developed a method to detect the presence of these weak or “background” events that to date have gone unnoticed, without having to detect each one individually. The method—which is currently being test driven by the LIGO community—” means that we may be able to look more than 8 billion light years further than we are currently observing,” Dr. Smith said.

“This will give us a snapshot of what the early universe looked like while providing insights into the evolution of the universe.”

A new cosmic evolution-based calculation that say that there are likely to be more than 36 ongoing intelligent civilizations throughout our Milky Way galaxy.

For the first time, a complete time-calibrated phylogeny for a large group of invertebrates is published for an entire continent.

In a recent research paper in the open-access, peer-reviewed academic journal ZooKeys, a German-Swedish team of scientists provide a diagrammatic hypothesis of the relationships and evolutionary history for all 496 European species of butterflies currently in existence. Their study provides an important tool for evolutionary and ecological research, meant for the use of insect and ecosystem conservation.

In order to analyse the ancestral relationships and history of evolutionary divergence of all European butterflies currently inhabiting the Old continent, the team led by Martin Wiemers—affiliated with both the Senckenberg German Entomological Institute and the Helmholtz Centre for Environmental Research—UFZ, mainly used molecular data from already published sources available from NCBI GenBank, but also contributed many new sequences, some from very local endemics for which no molecular data had previously been available.

Please listen to the second episode of my new Cosmic Controversy Podcast. This week’s guest is planetary scientist Stephen Kane at the University of California, Riverside, who discusses why Venus is so haunting and beguiling all at once.

In this wide-ranging interview, planetary scientist Stephen Kane of the University of California, Riverside, delves into the mysteries of our neighbor planet Venus. We discuss how Venus went wrong and why understanding its evolution is so important in characterizing extrasolar planetary systems like our own.

Every second of every day, countless biochemical reactions take place in our bodies’ cells. The organization of this complex system is the result of billions of years of evolution, fine-tuning our functions since the first primordial organisms.

One such vital reaction is “methylation,” where a methyl group —a carbon atom linked to three hydrogen atoms—attaches itself to a target molecule. Methylation is involved in the regulation of everything from DNA to proteins, and it is so vital that it can be found in all living organisms.

In a recent paper published in Communications Biology, a team of researchers lead by Jean-Michel Fustin and Hitoshi Okamura from Kyoto University’s Graduate School of Pharmaceutical Sciences has uncovered an intimate connection between methylation and the body’s circadian rhythms: a link that exists even in organisms that don’t traditionally “sleep,” such as bacteria.