In the fall, the clocks “fall back” one hour at 2:00 a.m., instantly becoming 1:00 a.m.
This week, we will lose an hour on Sunday March 9. We will gain an hour on Sunday November 2.
Daylight-saving time was originally concocted as a way to save energy in the evenings, and was implemented during World War I in Germany.
Deterioration of the hippocampus precedes and leads to memory impairment in late adulthood (1, 2). Strategies to fight hippocampal loss and protect against the development of memory impairment has become an important topic in recent years from both scientific and public health perspectives. Physical activity, such as aerobic exercise, has emerged as a promising low-cost treatment to improve neurocognitive function that is accessible to most adults and is not plagued by intolerable side effects often found with pharmaceutical treatments (3). Exercise enhances learning and improves retention, which is accompanied by increased cell proliferation and survival in the hippocampus of rodents (4– 6); effects that are mediated, in part, by increased production and secretion of BDNF and its receptor tyrosine kinase trkB (7, 8).
Aerobic exercise training increases gray and white matter volume in the prefrontal cortex (9) of older adults and increases the functioning of key nodes in the executive control network (10, 11). Greater amounts of physical activity are associated with sparing of prefrontal and temporal brain regions over a 9-y period, which reduces the risk for cognitive impairment (12). Further, hippocampal and medial temporal lobe volumes are larger in higher-fit older adults (13, 14), and larger hippocampal volumes mediate improvements in spatial memory (13). Exercise training increases cerebral blood volume (15) and perfusion of the hippocampus (16), but the extent to which exercise can modify the size of the hippocampus in late adulthood remains unknown.
To evaluate whether exercise training increases the size of the hippocampus and improves spatial memory, we designed a single-blind, randomized controlled trial in which adults were randomly assigned to receive either moderate-intensity aerobic exercise 3D/wk or stretching and toning exercises that served as a control. We predicted that 1 y of moderate-intensity exercise would increase the size of the hippocampus and that change in hippocampal volume would be associated with increased serum BDNF and improved memory function.
Summary: A new study finds that long-term use of non-steroidal anti-inflammatory drugs (NSAIDs) is associated with a lower risk of developing dementia. Researchers followed 11,745 adults over 14.5 years and found that those who used NSAIDs long-term had a 12% reduced dementia risk.
However, short-and intermediate-term NSAID use did not provide the same benefit, nor was the total cumulative dose linked to risk reduction. These findings suggest that sustained anti-inflammatory effects may play a role in protecting against dementia.
Past neuroscience studies suggest that memories of events that occurred at short time intervals from one another are often connected, via a process referred to as memory linking. While memory linking is now a well-documented phenomenon, its neural underpinnings have not been fully elucidated.
Researchers at the University of California Los Angeles (UCLA) recently carried out a study aimed at better understanding the neural processes that contribute to memory linking in the mouse brain. Their findings, published in Nature Neuroscience, suggest that dendritic plasticity, the adaptation of dendrites (i.e., branch-like extensions of neurons) over time, plays a key role in the linking of memories.
“A few years back, in a landmark study published in Nature in 2016, we demonstrated that memories formed a few hours apart are linked because they are stored in a common set of neurons in the hippocampus,” Alcino Silva, senior author of the paper, told Medical Xpress. “We wanted to know: Where within these neurons are these memories stored and linked? What was causing these neurons to be recruited?”
ETH Zurich researchers found that pupil size changes during sleep, indicating shifts in brain activity. This could help diagnose sleep and neurological disorders.
Our eyes are typically closed when we sleep. However, beneath our closed eyelids, a flurry of activity takes place. A team of researchers, led by principal investigators Caroline Lustenberger, Sarah Meissner, and Nicole Wenderoth from the Neural Control of Movement Lab at ETH Zurich, has observed that pupil size fluctuates constantly during sleep. Sometimes it increases, sometimes it decreases—sometimes these changes occur within seconds, while other times they unfold over several minutes.
We’ve combined lab-grown neurons with silicon chips and made it available to anyone, for first time ever.
“The Future Already Happened“
What if the past isn’t fixed? Scientists have just proven that the future can influence the past, shattering everything we thought we knew about time and reality. From mind-bending quantum experiments to the shocking science of precognition, this video explores the hidden connections between time, consciousness, and the universe.
✅GET YOUR FREE NUMEROLOGY READING HERE:
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Time Stamps:
0:00 — Mind-Blowing Experiments.
1:43 — Presentiment.
2:26 — Precognition.
5:12 — J.W. Dunne’s Precognitive Dream Protocol.
7:33 — Feeling The Future.
10:00 — Remote Viewing.
12:18 — Free Will & Retrocausality.
14:43 — Lucid Dreaming.
►Copyright ©:
Script — BE INSPIRED
Narration — BE INSPIRED
Footage is licensed through Videoblocks, Artgrid, and Envato.
Music: Epidemic Sound / Audiojungle / Envato Elements.
Interviews / Video References were used under FAIR USE LAW.
© BE INSPIRED CHANNEL — All rights reserved.
This is a collection of 68 philosophical sci-fi books recommended by you.
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After losing her family and freedom at the hands of the Imperial Network, Kathreen is sent to a brutal life of servitude, until a chance twist of fate grants her the identity of a powerful senator’s heiress. Determined to destroy the system that shattered her life, she vows to rise through their ranks—but to defeat them, she risks becoming a monster greater than any of them.
Rodolfo Llinas tells the story of how he has developed bundles of nanowires thinner than spider webs that can be inserted into the blood vessels of human brains.
While these wires have so far only been tested in animals, they prove that direct communication with the deep recesses of the brain may not be so far off. To understand just how big of a breakthrough this is—US agents from the National Security Agency quickly showed up at the MIT laboratory when the wires were being developed.
What does this mean for the future? It might be possible to stimulate the senses directly — creating visual perceptions, auditory perceptions, movements, and feelings. Deep brain stimulation could create the ultimate virtual reality. Not to mention, direct communication between man and machine or human brain to human brain could become a real possibility.
Llinas poses compelling questions about the potentials and ethics of his technology.
Newly discovered brain cells count each bite before sending the order to cease eating a meal. Columbia scientists have found specialized neurons in the brains of mice that order the animals to stop eating.
Though many feeding circuits in the brain are known to play a role in monitoring food intake, the neurons in those circuits do not make the final decision to cease eating a meal.
The neurons identified by the Columbia scientists, a new element of these circuits, are located in the brainstem, the oldest part of the vertebrate brain. Their discovery could lead to new treatments for obesity.
