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Inaugural Academy for Health and Lifespan Research (AHLR) …towards achieving the goal of not just accelerating longevity research, but making sure that its implications are accessible to all…”


The Academy is comprised of an elite group of world-renowned researchers, scientists and clinicians, all united toward making breakthroughs in longevity both accelerated & accessible.

Turn.bio is based on the scientific breakthrough work at Stanford of Vittorio Sebastiano, Jay Sarkar, and Marco Quarta. They are now leading the team to develop therapies that return mature differentiated cells to a dramatically younger state leaving their differentiated identity unaltered. Congrats!

More info on kizoo.com/en

Two important developers of the celebrated Crispr gene-editing technology said they will make it easier for researchers to license their intellectual property, a move aimed at hastening innovation in the burgeoning field.

MilliporeSigma, the life-sciences tools division of German pharmaceutical giant Merck KGaA, and the Broad Institute of MIT and Harvard said researchers will be able to get nonexclusive rights to patents held by both organizations for research purposes with a single license.

Companies will have to pay a licensing fee. Nonprofit and academic institutions will be permitted to license the patents for free, the organizations said in a news release on Thursday.

A team led by a Baylor University researcher has published a breakthrough article that provides a better understanding of the dynamic process by which sunlight-induced DNA damage is recognized by the molecular repair machinery in cells as needing repair.

Ultraviolet light from the sun is a ubiquitous carcinogen that can inflict structural damage to the cellular DNAs DNA carries important blueprints for cellular functions, failure in removing and restoring damaged parts of DNA in a timely fashion can have detrimental outcomes and lead to skin cancers in humans, said lead author Jung-Hyun Min, Ph.D., associate professor of chemistry and biochemistry in Baylor’s College of Arts & Sciences.

Min and her team showed how the repair Rad4/XPC would bind to one such UV-induced DNA damage—6–4 photoproduct—to mark the damaged site along the DNA in preparation for the rest of the nucleotide excision repair (NER) process in cells.