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To help with weight loss, many individuals turn to various strategies, including fad diets, intense exercise routines, and even supplements. However, one crucial factor often overlooked is the role of hormones in regulating appetite and metabolism. Glucagon-like peptide 1 (GLP-1) is a hormone that plays a significant role in satiety and glucose control. By increasing GLP-1 levels naturally, individuals can potentially enhance weight loss efforts (did you know Ozempic and Wegovy are not the only ways to do this!). Of course at STAT Wellness our goal is to uncover the root cause of your weight loss struggles; as a patient of STAT you will not get a blanket “you need to eat less and exercise more” mentality. So if the information in this article is not helpful, don’t settle until you have answers. We are complex beings and need to be treated that way. However the purpose of this article is to explore natural methods to boost GLP-1 levels and support weight loss efforts.

Consume Fiber-Rich Foods.

Dietary fiber has long been recognized as an essential component of a healthy diet, but its role in weight loss is often underestimated. Fiber-rich foods, such as whole grains, fruits, vegetables, and legumes, not only provide satiety but also help increase GLP-1 secretion. When consumed, fiber slows down digestion and promotes the release of GLP-1 from the intestinal cells. Additionally, certain soluble fibers act as prebiotics, nourishing beneficial gut bacteria, which further enhances GLP-1 production. Including a variety of fiber-rich foods in your diet can support weight loss efforts by promoting feelings of fullness and regulating appetite. Looking for ways to get more fiber consider: acacia fiber, ground flaxseed, chia seeds, avocado, raspberries, cruciferous vegetables, lentils, beans, and zen basil seeds (which are also lectin free).

NEW ORLEANS, March 18, 2024 — Doctors have long prescribed exercise to improve and protect health. In the future, a pill may offer some of the same benefits as exercise. Now, researchers report on new compounds that appear capable of mimicking the physical boost of working out — at least within rodent cells. This discovery could lead to a new way to treat muscle atrophy and other medical conditions in people, including heart failure and neurodegenerative disease.

The researchers will present their results today at the spring meeting of the American Chemical Society (ACS). ACS Spring 2024 is a hybrid meeting being held virtually and in person March 17–21; it features nearly 12,000 presentations on a range of science topics.

“We cannot replace exercise; exercise is important on all levels,” says Bahaa Elgendy, the project’s principal investigator who is presenting the work at the meeting. “If I can exercise, I should go ahead and get the physical activity. But there are so many cases in which a substitute is needed.”

Researchers from Weill Cornell Medicine have discovered that unique bacteria colonize the gut shortly after birth and make the neurotransmitter serotonin to educate gut immune cells that help in preventing allergic reactions to food and the bacteria themselves during early development.

The study published in the journal Science Immunology on March 15, 2024, revealed that bacteria abundant in the guts of newborns produce serotonin, which promotes the development of immune cells called T-regulatory cells or Tregs. These cells suppress inappropriate immune responses to help prevent autoimmune diseases and dangerous allergic reactions to harmless food items or beneficial gut microbes.

“The gut is now known as the second human brain as it makes over 90 percent of the neurotransmitters in the human body. While neurotransmitters such as serotonin are best known for their roles in brain health, receptors for neurotransmitters are located throughout the human body,” explained the study’s senior author, Dr. Melody Zeng, an assistant professor of immunology in the Gale and Ira Drukier Institute for Children’s Research and the Department of Pediatrics at Weill Cornell Medicine.

Researchers in the UC Davis Department of Biomedical Engineering are introducing a groundbreaking catheter-based device that could revolutionize heart attack and stroke prevention by enhancing intravascular imaging of dangerous plaques.


Researchers in the Department of Biomedical Engineering at the University of California, Davis, have developed a new catheter-based device that combines two powerful optical techniques to image the dangerous plaques that can build up inside the arteries that supply blood to the heart. By providing new details about plaque, the device could help clinicians and researchers improve treatments for preventing heart attacks and strokes.

Atherosclerosis occurs when fats, cholesterol and other substances accumulate on the artery walls, which can cause these vessels to become thick and stiff. A heart attack or stroke may occur if an atherosclerotic plaque inside the blood vessels ruptures or parts of it break off.

“Atherosclerosis, leading to heart attacks and strokes, is the number one cause of death in Western societies — exceeding all combined cancer types — and, therefore, a major public health issue,” said Laura Marcu, research team member leader and professor of biomedical engineering at UC Davis. “Better clinical management made possible by advanced intravascular imaging tools will benefit patients by providing more accurate information to help cardiologists tailor treatment or by supporting the development of new therapies.”

A new study provided light on the role of the protein STAP-1 in activating certain immune cells. Understanding STAP-1’s involvement in these cells may help researchers gain a better understanding of immune-related diseases and potential treatments.

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The researchers discovered that STAP-1 plays a key role in the activation of T cells, which are white blood cells that help the body defend itself against infections and preserve overall health. T cells are capable of identifying foreign substances that elicit an immune response (antigens) and developing tailored responses to destroy pathogens like bacteria and viruses.

Mercedes-Benz has officially employed humanoid robots to complete manual tasks on its assembly line. Apptronik’s Apollo robot will complete simple tasks like fetching and carrying, helping lighten the load for its human colleagues.

Announced on Friday, March 15, the robots will also be employed to complete other “low skill” tasks like basic assembly. This will free up human workers to focus on more important tasks and reduce the risk of repetitive strain injuries and other health and safety issues related to manual tasks.