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Sound-Powered Sensors stand to Save Millions of Batteries

Sensors that monitor infrastructure, such as bridges or buildings, or are used in medical devices, such as prostheses for the deaf, require a constant supply of power. The energy for this usually comes from batteries, which are replaced as soon as they are empty. This creates a huge waste problem. An EU study forecasts that in 2025, 78 million batteries will end up in the rubbish every day.

A new type of mechanical sensor, developed by researchers led by Marc Serra-Garcia and ETH geophysics professor Johan Robertsson, could now provide a remedy. Its creators have already applied for a patent for their invention and have now presented the principle in the journal Advanced Functional Materials.

Certain sound waves cause the sensor to vibrate “The sensor works purely mechanically and doesn’t require an external energy source. It simply utilizes the vibrational energy contained in sound waves,” Robertsson says.

How Obesity Dismantles our Mitochondria: Study reveals Key Mechanism behind Obesity-related Metabolic Dysfunction

The number of people with obesity has nearly tripled since 1975, resulting in a worldwide epidemic. While lifestyle factors like diet and exercise play a role in the development and progression of obesity, scientists have come to understand that obesity is also associated with intrinsic metabolic abnormalities.

Now, researchers from University of California San Diego School of Medicine have shed new light on how obesity affects our mitochondria, the all-important energy-producing structures of our cells.

In a study published in Nature Metabolism, the researchers found that when mice were fed a high-fat diet, mitochondria within their fat cells broke apart into smaller mitochondria with reduced capacity for burning fat. Further, they discovered that this process is controlled by a single gene. By deleting this gene from the mice, they were able to protect them from excess weight gain, even when they ate the same high-fat diet as other mice.

3D-Printed Electronic Skin provides promise for Human-Machine Interaction

With more than 1,000 nerve endings, human skin is the brain’s largest sensory connection to the outside world, providing a wealth of feedback through touch, temperature and pressure. While these complex features make skin a vital organ, they also make it a challenge to replicate.

By utilizing nanoengineered hydrogels that exhibit tunable electronic and thermal biosensing capabilities, researchers at Texas A&M University have developed a 3D-printed electronic skin (E-skin) that can flex, stretch and sense like human skin.

“The ability to replicate the sense of touch and integrate it into various technologies opens up new possibilities for human-machine interaction and advanced sensory experiences,” said Dr. Akhilesh Gaharwar, professor and director of research for the Department of Biomedical Engineering. “It can potentially revolutionize industries and improve the quality of life for individuals with disabilities.”

This new 3D printing method could solve the organ transplantation crisis

In the United States, the shortage of available organs for transplantation remains a critical issue, with over 100,000 individuals currently on the waiting list. The demand for organs, including hearts, kidneys, and livers, significantly outweighs the available supply, leading to prolonged waiting times and often, devastating consequences.

It is estimated that approximately 6,000 Americans lose their lives while waiting for a suitable donor organ every year.

Researchers at Carnegie Mellon University have developed a novel tissue engineering technique that aims to potentially bridge the gap between organ demand and availability, offering a beacon of hope.

Researchers discover that blocking ephrin B2 signaling can stop multiple myeloma growth

Cedars-Sinai Cancer investigators have discovered a protein expressed on multiple myeloma cancer cells that drives disease growth and development. The new study found that blocking part of the protein’s unique signaling pathway stops myeloma growth in culture and in laboratory mice. Their study was published in the journal Cancer Research.

The protein studied, called ephrin B2, is a powerful new target in the treatment of patients with multiple myeloma, a disease that has numerous partially effective treatments, but no cure. Based on these findings, investigators are now working on the development of therapies to target this protein in patients.

Myeloma cells grow inside a patient’s bone marrow. Unlike many types of cancer cells, multiple myeloma cells cannot live outside the patient, meaning they rely on signals from the patient’s healthy cells in order to grow. Investigators sought to determine the source of that signal as a potential way to block myeloma cells’ growth.

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