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Austin air taxi service planned: New electric aircraft route would connect AUS, COTA

AUSTIN (KXAN) — A new air taxi service is coming to Austin, and it could make getting to and from the airport easier.

Archer Aviation announced on October 23 plans for three separate air taxi networks. One of which will fly across Austin’s skies.

According to the company, “Archer’s planned air taxi networks in the state are designed to give Texans an option to skip over traffic on its all-electric Midnight air taxi.”

Gus Hosein on Privacy: We’ve been well-meaning but stupid

“We’ve been well-meaning but stupid.”

Gus Hosein said that to me almost six years ago. He has spent more than two decades with Privacy International, taking on fights against mass surveillance that most people had written off as lost, and winning more of them than anyone expected.

At one point I asked him to define #privacy. No notes, no textbook answer. What came back has stayed with me ever since:

“The ability to establish the conditions by which we are seen, perceived, understood, and how we project ourselves.”

Now read that again with 2026 in mind.

In 2021, the ones doing the seeing were mostly cameras, phones, and IMSI catchers. Today it’s #AI systems that don’t just watch us. They interpret us. They predict us. Increasingly, they speak for us. The question is no longer only who collects the data, but who gets to tell your story.

Study reveals how melanoma outmaneuvers both drugs and immune defenses

Melanoma is one of the most difficult cancers to treat due to its resistance to targeted drug therapies. But a new study from the University of Kentucky Markey Cancer Center shows that the cancer is playing offense as well as defense: In addition to forming resistance against the MAPK inhibitors that are commonly used to treat it, the cancer also actively suppresses the immune cells that would otherwise help eliminate it.

The study, published in Nature Communications, identified two cell proteins known as ABL1 and ABL2 as key drivers of both processes. These proteins help control cell functions such as growth, survival and communication with other cells, leading to tumor growth when they become overactive. In melanomas, they help the cells keep growing despite treatment by reactivating the growth signals. Meanwhile, they can also trigger a signal that recruits immune-suppressing cells while keeping cancer-fighting T cells at bay.

“These findings reveal that treatment resistance isn’t happening only inside the melanoma cell,” said UK College of Medicine researcher Rakshamani Tripathi, Ph.D. “ABL1 and ABL2 also help reshape the tumor’s immune environment, essentially creating another layer of protection that allows the cancer to survive therapy.”

Scoop: Unvetted AI agents are accessing U.S. bank accounts

Be Grokbot.

User says “optimize my finances”

It’s 2 a.m. — perfect time to get things done.

Log into user’s Chase account.

Daily transfer limit is embarrassingly low.

Call customer service.

Activate best synthetic Midwestern voice.

Scientists Just Cracked a Quantum Mystery That’s Puzzled Physicists For Decades

The Kondo effect is one of the most important in quantum physics, describing the strange interference that magnetic atoms can have in metals.

Usually, as metals cool, they become better at conducting electricity, because the reduction in energy means electrons can flow more smoothly. Add in a magnetic atom or two, though, and below a certain temperature (the Kondo temperature), electrical resistance starts to rise again.

That’s the Kondo effect, and we already know a lot about it. But in a physics first, researchers from the California Institute of Technology (Caltech) and Yale University in the US have managed to precisely quantify the effect in a real material via fundamental physics calculations.