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Researchers at Cornell University in the US have created wirelessly powered walking robot bugs that are tiny enough to be injected through an ordinary hypodermic needle.

The microscopic robots, which are each just 70 microns long, were produced using a multistep nanofabrication technique that turns a 4-inch specialised silicon wafer into a million microscopic robots in just weeks.

“The really high-level explanation of how we make them is we’re taking technology developed by the semiconductor industry and using it to make tiny robots,” explained Marc Miskin, assistant professor at the University of Pennsylvania, who developed the techniques whilst a post-doc at Cornell University with his colleagues professors Itai Cohen and Paul McEuen and researcher Alejandro Cortese.

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A look at the upcoming Undoing Aging Conference on March 28-30th, Berlin, Germany and some musings on why you should consider going.


As Undoing Aging 2019 in Berlin approaches, I have been reflecting on the previous year and the things that I am most looking forward to this year. So, today, as a journalist and longevity advocate, I am going to be sharing some of my personal musings and thoughts about this important event with you.

First of all, for those of you unfamiliar with the event, a little explanation is in order. Undoing Aging is an industry conference on March 28–30, Berlin, Germany that is organized by the SENS Research Foundation and the Forever Healthy Foundation.

The event has a firm focus on the science and progress being made in aging research and will see many of the leading experts come together for three days of exciting scientific talks, panels, and discussions. Undoing Aging 2019 is the second conference in this series and is one of the must-go events of the year.

Presents its list of the top 30 Longevity Influencers in Singapore, whose efforts in science, technology, industry and policy are driving the growth of the Singaporean Longevity Landscape.

Link to the Report: https://www.aginganalytics.com/longevity-in-singapore

Dr Finian Tan Brian Kennedy #DannySoon #GaryKhoo #BussarawanTeerawichitchainan #ChongHockSia #JaniceChia #JeffreyLu #CarlFirth #KanwaljitSoin #ChristianiJeyakumarHenry #ColinStewart #HweePinkTan Kenneth Noonan MD #LimChweeTeck #LokSheeMei #MelisTay #NeoKahYean #NgHuckHui #PaoloRampichini #PaulSi #PennyWan #JudithSwain #VishalDoshi #WallaceToores #WilfBlackburn #YuCai #LimXinhong WanJin Hong Birgit Lane.

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It won’t be simple. As with the advent of the car, many serious implications will be emergent, and the harshest effects borne by communities with the least powerful voices. We need to move our gaze from individuals to systems to communities, and back again. We must bring together diverse expertise, including workers and citizens, to develop a framework that health systems can use to anticipate and address issues. This framework needs an explicit mandate to consider and anticipate the social consequences of AI — and to keep watch over its effects. That is the best way to ensure that health technologies meet the needs of all, and not just those in Silicon Valley.


Health authorities are overlooking risks to systems and society in their evaluations of new digital technologies, says Melanie Smallman.

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There’s no better example of that than a 2016 discovery at the University of California, Irvine, by doctoral student Mya Le Thai. After playing around in the lab, she made a discovery that could lead to a rechargeable battery that could last up to 400 years. That means longer-lasting laptops and smartphones and fewer lithium ion batteries piling up in landfills.

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Join S-Booster 2019!


We will invite space-based business ideas from the Asia and Oceania regions!The Contest facilitates the realization of each business idea through open innovation with Japanese companies, and also provides financial support, business coaching and other assistances. Promoting Win-Win Business Cooperation Between Asia and Japan!

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Scientists have shown that the removal of non-dividing senescent cells, which are normally associated with aging, also appears to prevent Type 1 diabetes in diabetic mouse strains.

Clearing senescent beta cells prevents T1 diabetes

Type 1 diabetes (T1D) is a chronic condition in which the pancreas produces little or no insulin. Insulin is a hormone that allows sugar (glucose) to enter cells in order to create energy, so it is critical to cellular function and life.

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