These limping AI models could be a medical breakthrough đź
Via Seeker
How do you make âenvironmentally friendly carsâ by destroying the environment. This is really uncool. âWhat would Greta Do?â Environment activists had managed to halt the felling of the trees two days ago, arguing that Teslaâs Gigafactory would affect local wildlife and water supply. However Tesla is said to plant 3 trees for every one it cuts down. Is that good for the wildlife in the forest and the water table?
A high court in Berlin has allowed Tesla to continue clearing a 92 hectare forest for its massive Gigafactory. The court rejected pleas from environment activists and said that its decision was final.
Is our Universe really the only one? A new theory that hopes to solve one of the biggest problems in physics, may have rewritten our perception of time, and found a way through the Big Bang. Video by Howard Timberlake.
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A new international study confirms an association between a Mediterranean-type diet and better gut and systemic health later in life.
If this can be done, it is a game changer. Too much medicine treats instead of cures. Scientists at the Icahn School of Medicine at Mount Sinai along with collaborators study result is an important step toward a diabetes treatment that restores the bodyâs ability to produce insulin, according to the team.
Scientists at the Icahn School of Medicine at Mount Sinai along with collaborators from other institutions say they have discovered a novel combination of two classes of drugs that, together, cause the highest rate of proliferation ever observed in adult human ÎČ cells without harming most other cells in the body. The result is an important step toward a diabetes treatment that restores the bodyâs ability to produce insulin, according to the team.
The finding involved a DYRK1A inhibitor, which is known to cause ÎČ cells to proliferate, and a GLP1R agonist, already in widespread use in people with diabetes. Together, they caused the cells to proliferate at a rate of 5â6% per day. The teamâs research (âGLP-1 receptor agonists synergize with DYRK1A inhibitors to potentiate functional human beta cell regenerationâ) appears in Science Translational Medicine online.
Security of embedded devices is essential in todayâs internet-connected world. Security is typically guaranteed mathematically using a small secret key to encrypt the private messages.
When these computationally secure encryption algorithms are implemented on a physical hardware, they leak critical side-channel information in the form of power consumption or electromagnetic radiation. Now, Purdue University innovators have developed technology to kill the problem at the source itselfâtackling physical-layer vulnerabilities with physical-layer solutions.
Recent attacks have shown that such side-channel attacks can happen in just a few minutes from a short distance away. Recently, these attacks were used in the counterfeiting of e-cigarette batteries by stealing the secret encryption keys from authentic batteries to gain market share.
China is deploying robots and drones to remotely disinfect hospitals, deliver food and enforce quarantine restrictions as part of the effort to fight coronavirus.
Chinese state media has reported that drones and robots are being used by the government to cut the risk of person-to-person transmission of the disease.
There are 780 million people that are on some form of residential lockdown in China. Wuhan, the city where the viral outbreak began, has been sealed off from the outside world for weeks.
Now, five years later, their gamble appears to have paid off. Not only did New Horizons achieve a next-to flawless flyby of Arrokoth, the most distant object ever visited, but buried in its gigabytes of dataâwhich have been trickling back to Earth ever since the New Yearâs Day 2019 rendezvousâlies empirical evidence that strikes against a classic theory of how planets form. The New Horizons team published their latest analysis of the ancient body and how it came to be in a trio of papers appearing in Science last week.
Kingâs College in London, UK has been awarded a grant to investigate the role of senescent cells, which accumulate as we age, in the context of the heart and how using a therapy to remove them influences its ability to recover from injury.
What are senescent cells?
As you age, increasing numbers of your cells enter into a state known as senescence. Senescent cells do not divide or support the tissues of which they are part; instead, they emit a range of potentially harmful chemical signals that encourage nearby healthy cells to enter the same senescent state. Their presence causes many problems: they reduce tissue repair, increase chronic inflammation, and can even eventually raise the risk of cancer and other age-related diseases.