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MOSCOW (Reuters) — The International Space Station is leaking air in above-normal volumes, but the leak presents no danger to the Russian-American crew, the Russian space agency Roscosmos said on Tuesday.

The leak has been localised to one section of a service module and the crew, made up of U.S. astronaut Chris Cassidy and Russian cosmonauts Anatoly Ivanishin and Ivan Vagner, plan to eliminate it in the coming days, Russia’s RIA news agency quoted Roscosmos executive director Sergei Krikalev as saying.

Roscosmos said additional air may be delivered to the station.

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A team of researchers at MIT and other institutions say their “SPARC” compact fusion reactor should actually work — at least in theory, as they argue in a series of recently released research papers.

In a total of seven papers penned by 47 researchers from 12 institutions, the team argues that no unexpected impediments or surprises have shown up during the planning stages.

In other words, the research “confirms that the design we’re working on is very likely to work,” Martin Greenwald, deputy director of MIT’s Plasma Science and Fusion Center and project lead, told The New York Times.

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D-Wave today launched its next-generation quantum computing platform available via its Leap quantum cloud service. The company calls Advantage “the first quantum computer built for business.” In that vein, D-Wave today also debuted Launch, a jump-start program for businesses that want to begin building hybrid quantum applications.

“The Advantage quantum computer is the first quantum computer designed and developed from the ground up to support business applications,” D-Wave CEO Alan Baratz told VentureBeat. “We engineered it to be able to deal with large, complex commercial applications and to be able to support the running of those applications in production environments. There is no other quantum computer anywhere in the world that can solve problems at the scale and complexity that this quantum computer can solve problems. It really is the only one that you can run real business applications on. The other quantum computers are primarily prototypes. You can do experimentation, run small proofs of concept, but none of them can support applications at the scale that we can.”

D-Wave Systems’ Leap 2 is a quantum cloud service to help businesses and developers build and deploy quantum computing applications.

Continue reading “D-Wave announces Leap 2, its cloud service for quantum computing applications” | >

Not too much here, but longevity research fans might like.

Time may be our worst enemy, and aging its most powerful weapon. Our hair turns gray, our strength wanes, and a slew of age-related diseases represent what is happening at the cellular and molecular levels. Aging affects all the cells in our body’s different tissues, and understanding its impact would be of great value in fighting this eternal enemy of all ephemeral life forms.

The key is to first observe and measure. In a paper published in Cell Reports, scientists led by Johan Auwerx at EPFL started by asking a simple question: how do the tissues of aging mice differ from those of mice that are mere adults?

To answer the question, the researchers used the multiple techniques to measure the expression of everyone one of the thousands of mouse’s genes, and to identify any underlying epigenetic differences. The researchers not only measured different layers of information, but they did it across three different tissues: liver, heart, and muscle.

Continue reading “Understanding the effect of aging on the genome” | >

Stopping the cannibalistic behavior of a well-studied enzyme could be the key to new drugs to fight age-related diseases, according to a new study published online in Nature Cell Biology. For the first time, researchers in the Perelman School of Medicine at the University of Pennsylvania show how the self-eating cellular process known as autophagy is causing the SIRT1 enzyme, long known to play a role in longevity, to degrade over time in cells and tissue in mice. Identifying an enzymatic target is an important step that may lead to new or modified existing therapeutics.

“Blocking this pathway could be another potential approach to restore the level of SIRT1 in patients to help treat or prevent age-related organ and immune system decline,” said first author Lu Wang, Ph.D., a postdoctoral researcher in the lab of Shelly Berger, Ph.D., a professor of Cell and Developmental Biology in the Perelman School of Medicine and a professor of Biology in the School of Arts and Sciences at Penn. Berger also serves as senior author on the paper.

“The findings may be of most interest to the immune aging field, as autophagy’s role in SIRT1 in immune is a concept that hasn’t been shown before,” Wang added. “Exploiting this mechanism presents us with a new possibility of restoring immune function.”

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A newly identified genetic factor allows adult skin to repair itself like the skin of a newborn babe. The discovery by Washington State University researchers has implications for better skin wound treatment as well as preventing some of the aging process in skin.

In a study, published in the journal eLife on Sept. 29, the researchers identified a factor that acts like a molecular switch in the of baby mice that controls the formation of hair follicles as they develop during the first week of life. The switch is mostly turned off after skin forms and remains off in adult tissue. When it was activated in specialized cells in adult mice, their skin was able to heal wounds without scarring. The reformed skin even included fur and could make goose bumps, an ability that is lost in adult human scars.

“We were able to take the innate ability of young, neonatal skin to regenerate and transfer that ability to old skin,” said Driskell, an assistant professor in WSU’s School of Molecular Biosciences. “We have shown in principle that this kind of regeneration is possible.”

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Additionally, some reports have suggested that then-President Woodrow Wilson downplayed the virus, but that is a “wrong and a false trope of popular history,” Markel said. Wilson, who would later contract the virus, was organizing and commanding the U.S. effort in World War I and once the war ended, he sailed for Paris, where he stayed until April of 1919 organizing a peace treaty and the League of Nations, Markel said.

“The federal government played a very small role in American public health during that era. It was primarily a city and state role and those agencies were hardly downplaying it,” he said.

Unlike today, there was no CDC or national public health department. The Food and Drug Administration existed but consisted of a very small group of men. Additionally, there were no antibiotics, intensive care units, ventilators, IV fluids or vaccines. “You got a bed or maybe nursing care,” Markel said.

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