Lifeboat Foundation

Researchers have decoded the genomic sequence of Zygnema algae, revealing insights into the evolutionary transition from aquatic to terrestrial plant life. This breakthrough enhances our understanding of plant adaptation mechanisms and offers a basis for future studies in environmental resilience and bioenergy.

Plant life first emerged on land about 550 million years ago, and an international research team co-led by University of Nebraska–Lincoln computational biologist Yanbin Yin has cracked the genomic code of its humble beginnings, which made possible all other terrestrial life on Earth, including humans.

The team — about 50 scientists in eight countries – has generated the first genomic sequence of four strains of Zygnema algae, the closest living relatives of land plants. Their findings shed light on the ability of plants to adjust to the environment and provide a rich basis for future research.

This belief is slightly paradoxical as we have zero evidence that aliens even exist. What’s more, given the vast distances between star systems, it seems odd we’d only learn about them from a visit. Evidence for aliens is more likely to come from signals from faraway planets.

In a paper accepted for publication in the Proceedings of the International Astronomical Union, I argue that the belief in alien visitors is no longer a quirk, but a widespread societal problem.

About 2,890 kilometres beneath our feet lies a gigantic ball of liquid metal: our planet’s core. Scientists like me use the seismic waves created by earthquakes as a kind of ultrasound to “see” the shape and structure of the core.

Using a new way of studying these waves, my colleague Xiaolong Ma and I have made a surprising discovery: there is a large donut-shaped region of the core around the Equator, a few hundred kilometres thick, where seismic waves travel about 2% slower than in the rest of the core.

We think this region contains more lighter elements such as silicon and oxygen, and may play a crucial role in the vast currents of liquid metal running through the core that generate Earth’s magnetic field. Our results are published today in Science Advances.

Cyberattackers leverage Google Sheets for command control in a global espionage campaign targeting 70+ organizations.

In the digital realm, secrets (API keys, private keys, username and password combos, etc.) are the keys to the kingdom. But what if those keys were accidentally left out in the open in the very tools we use to collaborate every day?

A Single Secret Can Wreak Havoc

Imagine this: It’s a typical Tuesday in June 2024. Your dev team is knee-deep in sprints, Jira tickets are flying, and Slack is buzzing with the usual mix of cat memes and code snippets. Little do you know, buried in this digital chatter is a ticking time bomb – a plaintext credential that gives unfettered access to your company’s crown jewels.

Eight vulnerabilities have been uncovered in Microsoft applications for macOS that an adversary could exploit to gain elevated privileges or access sensitive data by circumventing the operating system’s permissions-based model, which revolves around the Transparency, Consent, and Control (TCC) framework.

“If successful, the adversary could gain any privileges already granted to the affected Microsoft applications,” Cisco Talos said. “For example, the attacker could send emails from the user account without the user noticing, record audio clips, take pictures, or record videos without any user interaction.”

The shortcomings span various applications such as Outlook, Teams, Word, Excel PowerPoint, and OneNote.

US government warns of rising attacks on infrastructure using advanced tactics.

Researchers at The University of Texas MD Anderson Cancer Center have shown that therapeutically restoring ‘youthful’ levels of a specific subunit of the telomerase enzyme can significantly reduce the signs and symptoms of aging in preclinical models. If these findings are validated in clinical trials, they could have important therapeutic implications for age-related diseases such as Alzheimer’s, Parkinson’s, heart disease, and cancer.

The study, published in Cell, identified a small molecule compound that restores physiological levels of telomerase reverse transcriptase (TERT), which normally is repressed with the onset of aging. Maintenance of TERT levels in aged lab models reduced cellular senescence and tissue inflammation, spurred new neuron formation with improved memory, and enhanced neuromuscular function, which increased strength and coordination.

The researchers show that TERT functions not only to extend telomeres, but also acts as a transcription factor to affect the expression of many genes directing neurogenesis, learning and memory, cellular senescence, and inflammation.

Pat Gelsinger’s Intel woes continue.