Toggle light / dark theme

‘Sun and rain’ detail how nanoparticles can escape from plastic coatings into the environment

If the 1967 film “The Graduate” were remade today, Mr. McGuire’s famous advice to young Benjamin Braddock would probably be updated to “Plastics … with nanoparticles.” These days, the mechanical, electrical and durability properties of polymers—the class of materials that includes plastics—are often enhanced by adding miniature particles (smaller than 100 nanometers or billionths of a meter) made of elements such as silicon or silver. But could those nanoparticles be released into the environment after the polymers are exposed to years of sun and water—and if so, what might be the health and ecological consequences?

In a recently published paper, researchers from the National Institute of Standards and Technology (NIST) describe how they subjected a commercial nanoparticle-infused coating to NIST-developed methods for accelerating the effects of weathering from ultraviolet (UV) radiation and simulated washings of rainwater. Their results indicate that humidity and exposure time are contributing factors for nanoparticle release, findings that may be useful in designing future studies to determine potential impacts.

In their recent experiment, the researchers exposed multiple samples of a commercially available polyurethane coating containing silicon dioxide nanoparticles to intense UV radiation for 100 days inside the NIST SPHERE (Simulated Photodegradation via High-Energy Radiant Exposure), a hollow, 2-meter (7-foot) diameter black aluminum chamber lined with highly UV reflective material that bears a casual resemblance to the Death Star in the film “Star Wars.” For this study, one day in the SPHERE was equivalent to 10 to 15 days outdoors. All samples were weathered at a constant temperature of 50 degrees Celsius (122 degrees Fahrenheit) with one group done in extremely dry conditions (approximately 0 percent humidity) and the other in humid conditions (75 percent humidity).

Nanosensors help understand how tumors will respond to therapies

MIT researchers have designed nanosensors that can profile tumors and may yield insight into how they will respond to certain therapies. The system is based on levels of enzymes called proteases, which cancer cells use to remodel their surroundings.

Once adapted for humans, this type of sensor could be used to determine how aggressive a tumor is and help doctors choose the best treatment, says Sangeeta Bhatia, the John and Dorothy Wilson Professor of Health Sciences and Technology and Electrical Engineering and Computer Science and a member of MIT’s Koch Institute for Integrative Cancer Research.

“This approach is exciting because people are developing therapies that are protease-activated,” Bhatia says. “Ideally you’d like to be able to stratify patients based on their protease activity and identify which ones would be good candidates for these therapies.”

Fund Medical Research to End Age-Related Disease

Please sign this petition to the NIH to help get more funding for aging research.


Every year about two million Americans die of illnesses doctors cannot cure. Cancer afflicts 50% of men and 30% of women. Five hundred and ninety five thousand Americans will die of cancer this year. Millions get heart diseases, strokes, etc. Every year 1,612,552 Americans die of the top 8 illnesses that doctors are unable to cure. Over a 30-year period, 48,376,560 United States citizens will die of the top 8 illnesses. Let us not forget other disabling and potentially curable illnesses. How much is it worth to save them? We have the resources and opportunity to cure age-related disease.

History has shown that medical research actually saves money. We now spend three trillion two hundred billion dollars yearly for health care. The health care expenditures will increase as our population grows with more senior citizens.

Every year we also spend hundreds of billions of dollars for services such as Social Security Disability, welfare, food stamps, special transportation, etc. Medical research will help cut down on the need for these services. It will also extend our lives.

Researchers May Have Found A Possible Cure For Crohn’s Disease

Crohn’s Disease is a legendarily difficult disease to not only identify but to treat or cure. The disease affects the intestines and digestive tract, stemming from bacteria in those areas building up and leading to serious side effects. It affects more than half a million people in the United States and is brutal – with the possibility of diarrhea, weight loss, fatigue, ulcers, malnutrition, and eventually colon cancer, liver disease or osteoporosis.

Treatment is limited, with the current best options being medicine to limit inflammation and prevent symptoms. There is no complete cure, only efforts to make life as comfortable and normal as possible for those afflicted. Without a specific target at which to aim some sort of treatment, the options for permanently reducing or removing symptoms and health risks are few and far between. Before now, the only thing scientists thought they knew about the disease’s cause was that E Coli was involved.

However, recent studies have led researches to believe that they have narrowed down other bacteria that contributes to the onset and ongoing symptoms of Crohn’s. Experts at the Center for Medical Mycology at Case Western Reserve and University Hospitals Cleveland Medical Center (wow, is that a mouthful!) think they have pinpointed two additional bacterial strains that contribute to the disease. They studied a cross section of people – those with the disease, those with the disease whose family members did not have it, and those without it – to attempt to identify common details in their biological tests. Such a diverse group of subjects is required not only due to the importance of adhering to the scientific method but because Crohn’s can be caused by genetics and environmental factors.

Mobile breath analyzer checks oral hygiene on the go

These days there are a quite a few high-tech ways to keep our oral hygiene in check, from toothbrushes that track your technique to smart floss dispensers that encourage healthy habits. Mint is the latest connected solution to hit bathrooms and beyond, and is said to detect signs of gum disease and poor oral hygiene on your breath in the space of a few seconds.

Developed by Breathometer, the same company behind the smartphone-based breathalyzer we covered back in 2013, Mint is small handheld device that hooks up with iOS and Android smartphones to check in on the state of affairs inside your mouth. After a successful Indiegogo campaign in March 2015 and some good attention at the CES conference that same year, the device has finally started shipping today.

A sensor array inside the device measures the volatile sulfur compounds (VSCs) in your breath. Studies have shown these to be key culprits behind bad breath, but their presence might do more than send your significant other running in the other direction. They can also be indicative of gum disease and poor oral health.

MIT: Powering up graphene implants without frying cells ~ For the Next Generation of Implants

This computational illustration shows a graphene network structure below a layer of water.

Image: Zhao Qin

New analysis finds way to safely conduct heat from graphene to biological tissues.

In the future, our health may be monitored and maintained by tiny sensors and drug dispensers, deployed within the body and made from graphene — one of the strongest, lightest materials in the world. Graphene is composed of a single sheet of carbon atoms, linked together like razor-thin chicken wire, and its properties may be tuned in countless ways, making it a versatile material for tiny, next-generation implants.

The science world is freaking out over this 25-year-old’s answer to antibiotic resistance

A 25-year-old student has just come up with a way to fight drug-resistant superbugs without antibiotics.

The new approach has so far only been tested in the lab and on mice, but it could offer a potential solution to antibiotic resistance, which is now getting so bad that the United Nations recently declared it a “fundamental threat” to global health.

Antibiotic-resistant bacteria already kill around 700,000 people each year, but a recent study suggests that number could rise to around 10 million by 2050.

A sickeningly bad idea indeed

A strong rebuttle to the sick article in the Telegraph which attempts to discredit Zuckerberg and Chan and their commitment to curing diseases.


Science and progress hardly ever stop just because a few cuckoos think we’re going too far. That’s what I tell myself most of the times when I bump into depressingly ill-informed articles about ageing and the diseases of old age. I tell myself that the best thing to do is to just let such articles disappear into oblivion and not give them any extra visibility. However, if instead of a few cuckoos we’re faced with an army of cuckoos, then we’re in for troubles.

At the time of this writing, people who are in favour of or oppose rejuvenation aren’t many, and neither are those who know about it but don’t care. Quite likely, most people in the world haven’t even heard about it yet. What I fear is that, when the advent of rejuvenation biotechnologies will be close, people who oppose rejuvenation will do their best to persuade undecided ones that disease is better than health, and ultimately, provoke an us-vs-them conflict that could jeopardise the cause of rejuvenation. The best way to avoid that conflict is to convince as many people as possible to support rejuvenation biotechnologies before they even arrive, so that when they do, those who oppose them will only be a few cuckoos indeed and not an army. Exposing the intellectual misery of deathist arguments is indubitably a good way of reaching this goal; that’s why I chose to respond to this spectacularly stupid article, instead of just ignoring it.

Lewis doesn’t want to live in a world without diseases. She prefers living in one where diseases are invented.

/* */