A new wearable patch can monitor your health through your sweat.
Fitness and health trackers are everywhere, but most of them are limited to collecting data on your heartbeat, how much your moving and information you manually input to their paired apps.
That’s helpful if you’re trying to get in shape, but for people suffering from chronic conditions and diseases — like kidney disease or cystic fibrosis — more exact and frequent analyses could alert them to life-endangering changes.
| Local | http://idahostatejournal.com/ Cutting calories (dieting) and increasing caloric expenditure (exercise) cause your brain to activate neurons that will not allow you to utilize fat or lose weight.
Recently, and at a most appropriate time, another study published in the journal eLife has given explanation as to why your current New Year’s Resolution diet will not work.
Cutting calories (dieting) and increasing caloric expenditure (exercise) cause your brain to activate neurons that will not allow you to utilize fat or lose weight.
Evolutionarily, when our ancestors living in what we now call Pocatello were hard pressed to find food, their brains stopped energy expenditure (using energy to move, be active, play, etc.) to slow them down to keep them alive.
More effective treatments for Multiple Sclerosis on the horizon?
Dr. Metcalfe and her company are already looking to take down other severe diseases as well.
“Psoriasis is high up on our list, and diabetes is another. Downstream there are all the dementias because a LIF is a major health factor for the brain. So if we can get it into the brain we can start protecting against dementia.”
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Microbes are often seen as pathogens that cause disease and antibiotics have been used successfully to combat these foreign invaders. In reality, the picture is more complex. Most of the time we live in harmony with our commensal gut microbiota, which is the collection of microorganisms colonizing the healthy gut. Commensal bacteria regulate host biological functions, including skeletal health. Researchers at the Medical University of South Carolina (MUSC) studying osteoimmunology, the interface of the skeletal and immune systems, have examined the impact of disrupting the healthy gut microbiome with antibiotics on post-pubertal skeletal development. Their results, published online on the January 16, 2019 in the American Journal of Pathology, showed that antibiotic disruption of the gut microbiota induced a pro-inflammatory response that led to increased activity of osteoclasts.
“This report introduces antibiotics as a critical exogenous modulator of gut microbiota osteoimmune response during post-pubertal skeletal development,” says Chad M. Novince, D.D.S., Ph.D., assistant professor in both the Colleges of Medicine and Dental Medicine who studies the impact of the microbiome on osteoimmunology and skeletal development. “People have shown that antibiotics perturb the microbiota, but this is the first known study evaluating how that has downstream effects on immune cells that regulate bone cells and the overall skeletal phenotype. This work brings the whole story together.”
The post-pubertal phase of development is a critical window of plasticity that supports the accrual of approximately 40 percent of our peak bone-mass. Recent work by the Novince lab and others has shown that the gut microbiota contributes to skeletal health. To determine the impact of antibiotic perturbation of the gut microbiota in post-pubertal skeletal development, Novince worked with team members at MUSC and treated mice with a cocktail of three antibiotics. In collaboration with microbiome scientist Alexander V. Alekseyenko, Ph.D., associate professor in the Biomedical Informatics Center and founding director of the MUSC Program for Human Microbiome Research, they were able to show that antibiotic treatment led to major alterations in the gut microbiota, resulting in specific changes to large groups of bacteria.
Wouldn’t it be great to power your home without having enormous costs to starting a journey on the alternative road? Now, you can achieve that and also take care of your figure! The founder of the Free Electric hybrid bike, Manoj Bhargava, says that his invention uses mechanical energy in the most basic way in order to transform an hour of exercise into supplying rural household with energy for 24 hours. The mechanism is simple: when you pedals, a flywheel is put in action, which turns the generator and thus charging a battery. What better motivation to work out from now on than to power your own home without any costs whatsoever? Watch the video featured to see the bike in action.
I’m excited to share my new article from Quartz on how science will make it safer and easier for a 50-year-old woman to have a child in 2028 than a 25-year-old woman today. #IVG and #DelayedFertilityAdvantage are game changers.
Women’s biological clocks drive human conception—and, in turn, human history.
Biology’s inflexible window of female fertility is generally agreed to be between the ages 18 and 35. Any older, and the risk of miscarrying, not getting pregnant at all, or bearing unhealthy children skyrockets. When the average lifespan for a woman in the Western world now hovers at around 80 years old, this means that less than 25% of her life can be spent easily (and safely) procreating.
Men have the luxury of being able sow their seed for most of their lives with few health ramifications (which is why someone like 72-year-old US president Donald Trump has a 12-year-old child). By comparison, the average woman will only ovulate 300 to 400 eggs in her lifetime, which means she only has the same amount of menstrual cycles to ever pursue procreation.
This seemingly unfair accident of human biology is all about to change, thanks to transhumanist science. Genetic editing combined with stem-cell technology will likely make it safer for a 50-year-old woman to have a baby in 2028 than for a 25-year-old woman in 2018. In two decades’ time, healthy 75-year-old women could be starting new families once more.
