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Aubrey De Grey and Reason from ’ Fight Aging’:


How can the current public focus on health be leveraged to promote a focus on prevention of disease, and aging as root cause for diseases?

Ole mensching, apollo ventures aubrey de grey, SENS research foundation sonia arrison, 100 plus capital reason, fightaging

TRANSCRIPT AND MP3: www.corbettreport.com/gates

Who is Bill Gates? A software developer? A businessman? A philanthropist? A global health expert? This question, once merely academic, is becoming a very real question for those who are beginning to realize that Gates’ unimaginable wealth has been used to gain control over every corner of the fields of public health, medical research and vaccine development. And now that we are presented with the very problem that Gates has been talking about for years, we will soon find that this software developer with no medical training is going to leverage that wealth into control over the fates of billions of people.

Deep Breathing alone will not create new lung cells, but combined with deep relaxation, guided visualizations, and therapeutic messages (Your stem cells are going to transform themselves into new lung cells to replace those lung cells infected by the Coronavirus. Your stem cells are going to change themselves into T-Cells, B-Cells, and Natural Killer Cells which are going to seek out, identify, attack, and destroy all the Coronavirus cells in your entire body), it will eliminate the coughing, fever, headaches, inflammation, and breathing problems.

While there is no hard evidence that lung exercises can help ease the discomfort and the progression of symptoms of COVID-19, there are techniques that will restore your lungs to optimal health.

Statistics is a useful tool for understanding the patterns in the world around us. But our intuition often lets us down when it comes to interpreting those patterns. In this series we look at some of the common mistakes we make and how to avoid them when thinking about statistics, probability and risk.

You don’t have to wait long to see a headline proclaiming that some food or behavior is associated with either an increased or a decreased health risk, or often both. How can it be that seemingly rigorous scientific studies can produce opposite conclusions?

Nowadays, researchers can access a wealth of software packages that can readily analyze data and output the results of complex statistical tests. While these are powerful resources, they also open the door to people without a full statistical understanding to misunderstand some of the subtleties within a dataset and to draw wildly incorrect conclusions.

Murphy’s Law: Everything that can go wrong will in fact go wrong.

Here is how to set the table for Murphy’s Law and become the epic center in the world for the COVID-19:

A. Eliminate the entire global health security team at the White House. Their job? Managing pandemics like COVID-19.

B. Misidentify the origin of COVID-19 as China, when the evidence says it came from France.

C. Check all Chinese at the Airport, while letting all Europeans enter the USA without being checked, bringing the COVID-19 virus in with them.

The breakthrough, which identified the location and function of every human gene, offered the promise of medical care tailored specifically to individual patients, based on their personal genetic makeup.

When researchers identified a gene associated with a 44 per cent risk of breast cancer in women, for example, it seemed that protecting them might be as simple as deactivating that gene.

But the promise of such personalized medicine has not fully materialized, say two McMaster researchers, because the full sophistication of the genetic blueprint has a more complex and far-reaching influence on human health than scientists had first realized.

Antibody found to block infection by the novel coronavirus SARS-CoV-2 in cells.

The ‘47D11’ antibody targets the ‘spike protein’ of the destructive coronavirus.

It could alter the ‘course of infection’ or protect an uninfected person exposed.

Here’s how to help people impacted by Covid-19.

The debate is focused on a subset of gain-of-function studies that manipulate deadly viruses to increase their transmissibility or virulence. “This is what happens to viruses in the wild”, explains Carrie Wolinetz, head of the NIH Office of Science Policy. “Gain-of-function experiments allow us to understand how pandemic viruses evolve, so that we can make predictions, develop countermeasures, and do disease surveillance”. Although none of the widely publicised mishaps of 2014 involved such work, the NIH decided to suspend funding for gain-of-function studies involving influenza, MERS-CoV, and SARS-CoV.


The US moratorium on gain-of-function experiments has been rescinded, but scientists are split over the benefits—and risks—of such studies. Talha Burki reports.

On Dec 19, 2017, the US National Institutes of Health (NIH) announced that they would resume funding gain-of-function experiments involving influenza, Middle East respiratory syndrome coronavirus, and severe acute respiratory syndrome coronavirus. A moratorium had been in place since October, 2014. At the time, the NIH had stated that the moratorium “will be effective until a robust and broad deliberative process is completed that results in the adoption of a new US Government gain-of-function research policy”. This process has now concluded. It was spearheaded by the National Science Advisory Board for Biosecurity (NSABB) and led to the development of a new framework for assessing funding decisions for research involving pathogens with enhanced pandemic potential. The release of the framework by the Department of Health and Human Services (HHS), of which NIH is part, signalled the end of the funding pause.

The situation has its roots in 2011, when the NSABB suppressed two studies involving H5N1 viruses that had been modified to allow airborne transmission from ferret to ferret. They worried that malign actors could replicate the work to deliberately cause an outbreak in human beings. After much debate, the studies were published in full in 2012. HHS subsequently issued guidelines for funding decisions on experiments likely to result in highly pathogenic H5N1 viruses transmissible from mammal to mammal via respiratory droplets. The guidelines were later expanded to include H7N9 viruses.

Maria became the very first COVID-19 patient to use Stem Cell Neurotherapy for COVID-19. In about 5 days, she will began to feel the healing effects of generating new lung cells which will eliminate her breathing problems.

We repurposed some tools from the Stem Cell Therapy for Cancer/Brain Tumor. Those tools are T-Cells, B-Cells, and Natural Killer Cells. Instead of programming those cancer killing cells to attack cancer cells, we have programmed them to seek out, identify, attack, and destroy all the Coronavirus cells in the entire body.

Stem Cell Neurotherapy sends therapeutic messages, e.g., “your stem cells are transforming into new cells for the lungs, liver, and kidneys” to the DNA inside the nucleus of stem cells. Inside the nucleus, the DNA receives the message and transmits it to the RNA, which translates the message into genetic code.

The genes inside the stem cells transmit the coded message to the proteins, which are converted by the mitochondria into ATP, which provides the energy for the coded message to transform the stem cells into a new set of lung cells, as well as new cells for the kidneys and liver.

These new cells in the lungs, kidneys, and liver will replace the cells that were infected by the COVID-19 virus. This results in the elimination of the coughing, fever, headaches, diarrhea, and breathing problems.