As Tesla is preparing to launch its ‘Tesla Semi’ electric truck next year, some are starting to imagine other possible applications than freight transport, like a Tesla Semi electric motorhome.
Motorhomes are often associated with freedom. The idea that you can take your entire home on the road and explore the world is extremely appealing to many.
Electric motorhomes would have the same appeal, but they could also push it to a whole new level.
Physicists from Nanyang Technological University, Singapore (NTU Singapore) and the Niels Bohr Institute in Copenhagen, Denmark, have devised a method to turn a non-magnetic metal into a magnet using laser light.
Magnets and their magnetic field are typically produced by circulating currents, like those found in everyday electromagnetic coils. The ‘handedness’ of these coils—whether they are wound in clockwise or anticlockwise fashion—determines the direction of the magnetic field produced.
The scientists theorise that when non-magnetic metallic disks are illuminated by linearly polarised light—light that does not possess any handedness of its own—circulating electric currents and hence magnetism can spontaneously emerge in the disk.
A research team lead by Osaka University demonstrated how information encoded in the circular polarization of a laser beam can be translated into the spin state of an electron in a quantum dot, each being a quantum bit and a quantum computer candidate. The achievement represents a major step towards a “quantum internet,” in which future computers can rapidly and securely send and receive quantum information.
Quantum computers have the potential to vastly outperform current systems because they work in a fundamentally different way. Instead of processing discrete ones and zeros, quantum information, whether stored in electron spins or transmitted by laser photons, can be in a superposition of multiple states simultaneously. Moreover, the states of two or more objects can become entangled, so that the status of one cannot be completely described without this other. Handling entangled states allow quantum computers to evaluate many possibilities simultaneously, as well as transmit information from place to place immune from eavesdropping.
However, these entangled states can be very fragile, lasting only microseconds before losing coherence. To realize the goal of a quantum internet, over which coherent light signals can relay quantum information, these signals must be able to interact with electron spins inside distant computers.
Researchers are still optimistic about finding disease-altering medicines—just not anytime soon.
The spinning ball of plasma that is our Sun produces a spinning magnetic field too, and where that magnetic field weakens, solar winds can escape.
Now scientists have been able to recreate those same effects in a lab for the first time, meaning we can study the bizarre science around our star at close quarters, without a trip across the Solar System.
Knowing how this magnetic field and its associated plasma flows behave is crucial in improving our understanding of how and when solar storms might impact Earth, and potentially put our communications systems and infrastructure under severe strain.
For the past quarter century, scientists battled Alzheimer’s disease under a single guiding principle: that protein clumps—beta-amyloid—deposited outside sensitive brain cells gradually damage neuronal functions and trigger memory loss. The solution seems simple: remove junk amyloid, protect the brain.
They could be completely wrong.
Last month, Alzheimer’s disease defeated another promising near-market drug that tried to prevent or remove amyloid deposits, adding to the disease’s therapeutic “graveyard of dreams.” Although the drug removed toxic amyloid, the patients didn’t get better. The failure is once again spurring scientists to confront an uncomfortable truth: targeting amyloid clumps when patients already show memory symptoms doesn’t work. Wiping out soluble amyloid—fragments of proteins before they aggregate into junk—also dead ends.
What if neither distance nor language mattered? What if technology could help you be anywhere you need to be and speak any language? Using AI technology and holographic experiences this is possible, and it is revolutionary.
Microsoft has created a hologram that will transform someone into a digital speaker of another language. The software giant unveiled the technology during a keynote at the Microsoft Inspire partner conference this morning in Las Vegas. Microsoft recently scanned Julia White, a company executive for Azure, at a Mixed Reality capture studio to transform her into an exact hologram replica.
The digital version appeared onstage to translate the keynote into Japanese. Microsoft has used its Azure AI technologies and neural text-to-speech to make this possible. It works by taking recordings of White’s voice, in order to create a personalized voice signature, to make it sound like she’s speaking Japanese.
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Mexico’s #Popocatepetl volcano spews gas and ash into the air. Some 25 million people live about 60 miles (100km) from the crater (Source: TicToc)
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Maybe you’ve gotten a bit blasé about rocket landings. SpaceX has pulled off more than 40 of them during orbital launches, after all, and Blue Origin has done it 10 times on test flights of its New Shepard suborbital vehicle.
The journal club hosted by Dr. Oliver Medvedik returns for July and takes a look at the new SIRT6 evolutionary biology paper by Dr. Vera Gorbunova and collaborators, showing a relationship between enhanced SIRT6 function and longevity.
Abstract DNA repair has been hypothesized to be a longevity determinant, but the evidence for it is based largely on accelerated aging phenotypes of DNA repair mutants. Here, using a panel of 18 rodent species with diverse lifespans, we show that more robust DNA double-strand break (DSB) repair, but not nucleotide excision repair (NER), coevolves with longevity. Evolution of NER, unlike DSB, is shaped primarily by sunlight exposure. We further show that the capacity of the SIRT6 protein to promote DSB repair accounts for a major part of the variation in DSB repair efficacy between short- and long-lived species. We dissected the molecular differences between a weak (mouse) and a strong (beaver) SIRT6 protein and identified five amino acid residues that are fully responsible for their differential activities. Our findings demonstrate that DSB repair and SIRT6 have been optimized during the evolution of longevity, which provides new targets for anti-aging interventions.
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