Lifeboat Foundation: Safeguarding Humanity
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Scientists have discovered hundreds of unusually large, bacteria-killing viruses with capabilities normally associated with living organisms, blurring the line between living microbes and viral machines.

These phages—short for bacteriophages, so-called because they “eat” bacteria—are of a size and complexity considered typical of life, carry numerous genes normally found in bacteria and use these genes against their bacterial hosts.

University of California, Berkeley, researchers and their collaborators found these huge phages by scouring a large database of DNA that they generated from nearly 30 different Earth environments, ranging from the guts of premature infants and pregnant women to a Tibetan hot spring, a South African bioreactor, hospital rooms, oceans, lakes and deep underground.

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Next week, the European Space Agency is going to jettison a cubesat called Qarman from the International Space Station and watch it burst into a fireball as it reenters Earth’s atmosphere—all on purpose.

What’s the mission: Qarman (short for “QubeSat for Aerothermodynamic Research and Measurements on Ablation”) is a shoebox-sized experiment meant to help researchers better understand the physics at play when objects plummet into the planet’s atmosphere and burn up. Qarman was brought up to the ISS in December during a cargo resupply mission. On February 17, it will be cast back out into space and begin slowly drifting toward Earth before entering the atmosphere and burning up in about six months.

Tell me more: Qarman has four solar-cell-covered panels that are designed to increase atmospheric drag and hasten reentry. Its nose is made from a special kind of cork that’s typically used in thermal protection systems on spacecraft. Ground testing shows that when the cork heats up, it chars and flakes away a bit at a time. The Qarman team is interested in learning how this process works during reentry.

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But researchers just located a baby giant exoplanet orbiting a young star just 330 light-years from Earth, making it the closest of its kind to us.

The planet is known as 2MASS 1155–7919 b, and it’s located in Epsilon Chamaeleontis Association, a young group of stars seen in our southern sky near the Chameleon constellation.

Researchers from the Rochester Institute of Technology made the discovery using data collected by the European Space Agency’s Gaia space observatory.

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Researchers in the Netherlands have developed an incredibly accurate nanosensor which can detect metastatic cancer cells from just a single drop of blood in a major breakthrough for early detection and treatment of the disease.

PhD students Dilu Mathew from University of Twente and Pepijn Beekman from Wageningen University pooled their resources and developed a tiny system to detect tumor-derived extracellular vesicles (tdEVs), a particular type of cancer biomarker.

Their nanosensor is so sensitive it can detect cancer biomarkers on a broad spectrum of concentrations from 10 particles per microliter to 1 million particles per microliter, thanks to its incredibly small and delicate electrodes, shaped like two combs facing each other, with a gap of just 120 nanometers between them.

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A molecular switch has been identified by scientists at the University of California that controls the immune machinery which is responsible for chronic inflammation within the body; findings published in the journal Cell Metabolism may lead to new ways to halt and/or reverse age related conditions such as cancer, diabetes, Alzheimer’s and Parkinson’s disease.

“My lab is very interested in understanding the reversibility of aging,” said senior author Danica Chen, associate professor of metabolic biology, nutritional sciences and toxicology at UC Berkeley. “In the past, we showed that aged stem cells can be rejuvenated. Now, we are asking: to what extent can aging be reversed? And we are doing that by looking at physiological conditions, like inflammation and insulin resistance, that have been associated with aging-related degeneration and diseases.”

A bulky collection of NLRP3 inflammasome immune proteins which are responsible for sensing potential threats to the body and launching an inflammatory response were shown to be essentially switched off by removing some molecular matter in a deacetylation process. Overactivation of NLRP3 inflammasomes is linked to a range of chronic conditions such as cancer, dementia, diabetes, and multiple sclerosis; this study suggests that drugs targeted towards deacetylation these NLRP3 inflammasomes may help to prevent and/or treat many age related conditions and even possibly age related degeneration itself in general.

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