Posted in space travel
Sachika Bhatia (Space Renaissance India) interviews.
Adriano V. Autino, President of Space Renaissance International.
Questioned about space privatization, Autino discusses “Space Exploration vs. Space Settlement”
During a webinar on Space Awareness organized by Space Renaissance India.
Dean Kamen, the inventor of the Segway, is currently spearheading a project to convert part of the old New Hampshire textile plant into a factory for lab-grown lungs, livers, and other organs for transplantation — and he doesn’t think it’ll take long to do it.
The nonprofit is like a club for tissue engineering and regenerative medicine researchers. Groups must have something to offer in order to join (money, equipment, experience), but once a part of ARMI, they gain access to the other members’ research and resources.
Posted in robotics/AI
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In Moscow, the Colonel’s recipe is now being served by robotic arms!
Observers said that the fully fledged system could put China in a competitive position against the world’s three other satellite navigation networks – the US-owned Global Positioning System (GPS), the European Union’s Galileo and Russia’s Glonass. It could also expand China’s international engagement and offer an alternative to other countries that had been reliant on US technology, they said.
Final satellite in the navigation network – China’s answer to the US-owned GPS – is sent into orbit on Tuesday after technical problems delayed it by a week.
Scientists identify an important protein that increases “bacterial virulence,” when mutated, changing harmless bacteria to harmful ones.
As far as humans are concerned, bacteria can be classified as either harmful, pathogenic bacteria and harmless or beneficial non-pathogenic bacteria. To develop better treatments for diseases caused by pathogenic bacteria, we need to have a good grasp on the mechanisms that cause some bacteria to be virulent. Scientists have identified genes that cause virulence, or capability to cause disease, but they do not fully know how bacteria evolve to become pathogenic.
To find out, Professor Chikara Kaito and his team of scientists from Okayama University, Japan, used a process called experimental evolution to identify molecular mechanisms that cells develop to gain useful traits, and published their findings in PLoS Pathogens. “We’re excited by this research because no one has ever looked at virulence evolution of bacteria in an animal; studies before us looked at the evolution in cells,” said Prof Kaito.
The Longevity Vision Fund invests in technologies, products, and services that extend healthy human lifespans and decrease negative aging effects.
Fat cell aspirste can be part of regenerative medicine treatments for degenerated body tissues such as joints, muscles, tendon and ligaments.
Three studies showing large DNA deletions and reshuffling heighten safety concerns about heritable genome editing.
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While there are ways to alleviate some symptoms, there is currently no way to prevent or cure Parkinson’s disease, so the prospect of a one-off treatment that completely eliminates it is certainly an exciting one. While such a therapy remains a while off, scientists have demonstrated an exciting proof of concept in mice, whereby inhibiting a single gene as a one-time treatment eradicated the disease entirely, and kept it at bay for the remainder of their lives.
The research was carried out at the University of California, San Diego (UCSD), and centers on a protein called PTB, which plays a role in which genes are switch on and off in a cell. The team was experimenting with techniques whereby the gene that encodes for PTB is switched off so researchers can determine the flow-on effects of a reduction in the that protein on other cell types, and found peculiar results when working with connective tissue cells called fibroblasts.
In one experiment, the team created a cell line that was permanently lacking PTB, and after a couple of weeks found that there was only a small amount of fibroblasts remaining in the dish, which was brimming with neurons instead. Building on this, the team was able to use a single treatment to inhibit the activity of PTB in mice, which reprogrammed support cells in the brain called astrocytes into neurons that produce the neurotransmitter dopamine.
