Lifeboat Foundation

It’s a reference to the evil form in the ‘Lord of the Rings’ books. For those unfamiliar with the ‘Lord of the Rings” books and movies, the Eye of Sauron is the chief antagonist in the series, exemplified as a flaming eye and that is a metaphor for pure evil. It’s not something anyone would want to be compared to unless, of course, you are Meta founder and CEO Mark Zuckerberg.

Mark Zuckerberg on Long-Term Strategy, Business and Parenting Principles, Personal Energy Management, Building the Metaverse, Seeking Awe, the Role of Religion, Solving Deep Technical Challenges (e.g., AR), and More | Brought to you by Eight Sleep’s Pod Pro Cover sleeping solution for dynamic cooling and heating (http://eightsleep.com/Tim), Magic Spoon delicious low-carb cereal (http://magicspoon.com/tim), and Helium 10 all-in-one software suite to sell on Amazon (https://helium10.com/tim).

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PX-478 improves insulin secretion in mouse and human organoid models of diabetes.

Captured CO₂ products include non-petroleum fuels, vodka, perfume, foam, insulation, mattresses, and carbon fibre for buildings and cars.

Innovations in computing tech have improved the accuracy of DNA synthesis and enabled synthetic biology to work in the real world.

I don’t know about you, but I’m constantly looking for the “next big thing” in the stock market. And I think synthetic biology might just be it.

Why? If you invested just $10,000 into any of those world-changing stocks back in their early days, you’d have MILLIONS today. Forget the Iraq War, the housing crash, the European debt crisis. Forget the pandemic and the Russia-Ukraine war. Through it all, you’d have millions today.

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BBC News.

Developers hope their ‘slime robot’ will one day be used to help find items that have been swallowed by accident.

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I did a short youtube video discussing the space arena of the war in Ukraine.

Space War, what is it good for?

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The discovery that short peptides can form spontaneously on cosmic dust hints at more of a role for them in the origin of life, on Earth or elsewhere.

A miniature review of.

The risk of morbidity and mortality increases exponentially with age. Chronic inflammation, accumulation of DNA damage, dysfunctional mitochondria, and increased senescent cell load are factors contributing to this. Mechanistic investigations have revealed specific pathways and processes which, proposedly, cause age-related phenotypes such as frailty, reduced physical resilience, and multi-morbidity. Among promising treatments alleviating the consequences of aging are caloric restriction and pharmacologically targeting longevity pathways such as the mechanistic target of rapamycin (mTOR), sirtuins, and anti-apoptotic pathways in senescent cells. Regulation of these pathways and processes has revealed significant health-and lifespan extending results in animal models. Nevertheless, it remains unclear if similar results translate to humans. A requirement of translation are the development of age-and morbidity associated biomarkers as longitudinal trials are difficult and not feasible, practical, nor ethical when human life span is the endpoint. Current biomarkers and the results of anti-aging intervention studies in humans will be covered within this paper. The future of clinical trials targeting aging may be phase 2 and 3 studies with larger populations if safety and tolerability of investigated medication continues not to be a hurdle for further investigations.

As age increases, so does the susceptibility to a series of chronic diseases which ultimately result in fatal endings. This is such a basic part of life that we rarely consider if there is anything we can do to postpone it. So far, the principal of “one-disease-one-treatment” has brought medical sciences far but this line of thought may soon be outdated when it comes to aging related conditions. It is like fighting a many-headed monster: If one condition is treated successfully, another emerges shortly after. This point is illustrated as eradicating the two leading causes of death (cancer and cardiovascular disease) extends mean life span by 3.3 and 4 years, respectively (Arias et al., 2013). Interestingly, the gain of treating multiple diseases combined exceeds the sum of these numbers.

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