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For decades, transistors—the heart of computer chips—have been getting smaller and smaller. As a result, the electronic components in many devices can be made even more compact, faster and also more powerful. But is this development coming to a natural halt? The smaller the components, the greater the danger that individual defects in the atomic structure will significantly change the behavior of the component. This applies to the established silicon technology and novel nanotechnologies based on 2D materials.

At Vienna University of Technology (TU Wien), intensive work has been done on the physical description of this problem at the transistor level. Now the researchers are going a step further and looking at the influence of defects at the level of electronic circuits, which sometimes consist of several—sometimes even billions—of transistors. In some cases, individual transistors can operate outside the desired specification, but still perform well as part of a circuit consisting of several transistors. With this new approach at the circuit level, significant advances in miniaturization are still possible.

The study is published in the journal Advanced Materials.

This video was recorded at the Foresight Vision Weekend 2022 at Château du Feÿ in France.

Michael Greve | Longevity Investing.

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An experimental campaign was conducted on the Säntis mountain in north-eastern Switzerland during the summer of 2021 with a high-repetition-rate terawatt laser. The guiding of an upward negative lightning leader over a distance of 50 m was recorded by two separate high-speed cameras.


A terawatt laser filament is shown to be able to guide lightning over a distance of 50 m in field trials on the Säntis mountain in the Swiss Alps.

Plants are often thought of as sources of food, oxygen, and decoration, but not as a source of electricity. However, scientists have discovered that by harnessing the natural transport of electrons within plant cells, it is possible to generate electricity as part of a green, biological solar cell. In a recent study published in ACS Applied Materials & Interfaces, researchers for the first time used a succulent plant to create a living “bio-solar cell” that runs on photosynthesis.

Photosynthesis is how plants and some microorganisms use sunlight to synthesize carbohydrates from carbon dioxide and water.

Researchers in Drexel University’s College of Engineering have developed a thin film device, fabricated by spray coating, that can block electromagnetic radiation with the flip of a switch. The breakthrough, enabled by versatile two-dimensional materials called MXenes, could adjust the performance of electronic devices, strengthen wireless connections and secure mobile communications against intrusion.

The team, led by Yury Gogotsi, Ph.D., Distinguished University and Bach professor in Drexel’s College of Engineering, previously demonstrated that the two-dimensional layered MXene materials, discovered just over a decade ago, when combined with an , can be turned into a potent active shield against .

This latest MXene discovery, reported in Nature Nanotechnology, shows how this shielding can be tuned when a small voltage—less than that produced by an alkaline battery—is applied.

Findings solve a 60-year-old problem, researchers say A new 10-minute scan could make way for the most common cause of high blood pressure to be detected and cured, new research has suggested. Using a new type of CT scan, doctors were able to cure high blood pressure by lighting up nodules (tiny growths) in a hormone gland cure and removing them.

The Space Solar Power Project (SSPP) began in 2011 when Donald Bren — philanthropist, chairman of the Irvine Company, and a lifetime member of the Caltech Board of Trustees — and Caltech’s then-president Jean-Lou Chameau came together to discuss the potential for a space-based solar power research project. By 2013, Bren and his wife (Caltech trustee Brigitte Bren) began funding the project through the Donald Bren Foundation, which will eventually exceed $100 million. As Bren said in a recent Caltech press release:

“For many years, I’ve dreamed about how space-based solar power could solve some of humanity’s most urgent challenges. Today, I’m thrilled to be supporting Caltech’s brilliant scientists as they race to make that dream a reality.”

While the technology behind solar cells has existed since the late 19th century, generating solar power in space presents some serious challenges. For one thing, solar panels are heavy and require extensive wiring to transmit power, making them expensive and difficult to launch. To overcome these challenges, the SSPP team had to create a satellite that would be light enough for cost-effective launches yet strong enough to withstand the extreme environment of space. This required envisioning and developing new technologies, architectures, materials, and structures.

Scientists at Harvard Medical School have investigated why we age, and identified a possible way to reverse it. In tests in mice, the team showed that epigenetic “software glitches” drive the symptoms of aging – and a system reboot can reverse them, potentially extending lifespan.

Our genome contains our complete DNA blueprint, which is found in every single cell of our bodies. But it’s not the whole picture – an extra layer of information, known as the epigenome, sits above that and controls which genes are switched on and off in different types of cells. It’s as though every cell in our body is working from the same operating manual (the genome), but the epigenome is like a table of contents that directs different cells to different chapters (genes). After all, lung cells need very different instructions to heart cells.

Environmental and lifestyle factors like diet, exercise and even childhood experiences could change epigenetic expression over our lifetimes. Epigenetic changes have been linked to the rate of biological aging, but whether they drove the symptoms of aging or were a symptom themselves remained unclear.

Chromatin structures and transcriptional networks are known to specify cell identity during development which directs cells into metaphorical valleys in the Waddington landscape. Cells must retain their identity through the preservation of epigenetic information and a state of low Shannon entropy for the maintenance of optimal function. Yeast studies in the 1990s have reported that a loss of epigenetic information compared to genetics can cause aging. Few other studies also confirmed that epigenetic changes are not just a biomarker but a cause of aging in yeasts.

Epigenetic changes associated with aging include changes in DNA methylation (DNAme) patterns, H3K27me3, H3K9me3, and H3K9me3. Many epigenetic changes have been observed to follow a specific pattern. However, the reason for changes in the mammalian epigenome is not yet known. A few clues can be obtained from yeast, where DSB is a significant factor whose repair requires epigenetic regulators Esa1, Gcn5, Rpd3, Hst1, and Sir2. As per the ‘‘RCM’’ hypothesis and ‘Information Theory of Aging’’, aging in eukaryotes occurs due to the loss of epigenetic information and transcriptional networks in response to cellular damage such as a crash injury or a DSB.

A new study in the journal Cell aimed to determine whether epigenetic changes are a cause of mammalian aging.