Lifeboat Foundation: Safeguarding Humanity
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Graphene is known as the world’s thinnest material due to its 2-D structure, in which each sheet is only one carbon atom thick, allowing each atom to engage in a chemical reaction from two sides. Graphene flakes can have a very large proportion of edge atoms, all of which have a particular chemical reactivity. In addition, chemically active voids created by missing atoms are a surface defect of graphene sheets. These structural defects and edges play a vital role in carbon chemistry and physics, as they alter the chemical reactivity of graphene. In fact, chemical reactions have repeatedly been shown to be favoured at these defect sites.

Interstellar molecular clouds are predominantly composed of hydrogen in molecular form (H2), but also contain a small percentage of dust particles mostly in the form of carbon nanostructures, called polyaromatic hydrocarbons (PAH). These clouds are often referred to as ‘star nurseries’ as their low temperature and high density allows gravity to locally condense matter in such a way that it initiates H fusion, the nuclear reaction at the heart of each star. Graphene-based materials, prepared from the exfoliation of graphite oxide, are used as a model of interstellar carbon dust as they contain a relatively large amount of , either at their edges or on their surface. These defects are thought to sustain the Eley-Rideal chemical reaction, which recombines two H into one H2 molecule.

The observation of interstellar clouds in inhospitable regions of space, including in the direct proximity of giant stars, poses the question of the origin of the stability of hydrogen in the molecular form (H2). This question stands because the clouds are constantly being washed out by intense radiation, hence cracking the hydrogen molecules into atoms. Astrochemists suggest that the chemical mechanism responsible for the recombination of atomic H into molecular H2 is catalysed by carbon flakes in interstellar clouds. Their theories are challenged by the need for a very efficient surface chemistry scenario to explain the observed equilibrium between dissociation and recombination. They had to introduce highly reactive sites into their models so that the capture of an atomic H nearby occurs without fail.

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After labor, not all of us will want to explore inner consciousness. Abundant leisure will not turn everyone into the Buddha. Many of our tastes are in the gutter, and I have no objection to leaving them there. I’m not a fan of shopping per se, but buying stuff is deeply satisfying and motivating for many people. Is it possible to rethink the pleasure of conspicuous consumption in a way that decouples it from the competitive labor economy? The post-work world I’m imagining will have little surplus money for unnecessary shopping, even if robots and computers can dramatically lower the overhead of such production. So, a non-consummatory form of shopping will have to be cultivated.

Some people marshal all their evolved predatory skills to hunt down the perfect sweater, shoes, or watch. Could we redesign shopping as a system of “catch-and-release,” so that, like sport fishing, it’s the adventure and not the prize that becomes central? Maybe we will hunt for luxury items, but then instead of keeping them, simply photograph ourselves wearing the items (like a fisherman holding a giant pike). It’s an unlikely adjustment, I’ll grant you, but I never thought catch-and-release fishing would be fun until I did it, and it was. The way some people already buy and return items suggests to me that catch-and-release shopping is not impossible.

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Daily Caller

China is making progress on its first indigenous aircraft carrier, the Shandong.

After two years and nine months of construction, China’s first domestically-built aircraft carrier is “taking shape.” The ship is under construction at a shipyard in Dalian, where the superstructure has already been mounted onto the hull. The vessel is expected to be launched this year; however, it will probably be a few more years before the ship enters military service.

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Judging by the way some users handle portable consumer electronics, it’s fair to say that they can be considered harsh environment devices. Cell phones, MP3 players, tablets and other portable electronic devices have become ubiquitous personal and professional tools that are used constantly throughout the day and not with the gentlest of care. As a result, switch manufacturers must create new rugged miniature switches that combine significant space and weight reductions with ruggedness and long operating lives.

These miniature switches must function in the same reliable, consistent manner as the more substantially-sized industrial design, all the while maintaining optimum functionality, performance and extended lifespans. Switch manufacturers that offer value-added services, including manufacturing modules and custom assemblies, can deliver complete electromechanical solutions that not only meet the size and performance requirements, but can also withstand the elements like vibration and shock.

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Brain resets during sleep; guess why I need my 5 shot espressos in the morning.

Summary: Researchers examine if the size of synapses alters during sleep and wake states.

Source: University of Wisconsin Madison.

Striking electron microscope pictures from inside the brains of mice suggest what happens in our own brain every day: Our synapses – the junctions between nerve cells – grow strong and large during the stimulation of daytime, then shrink by nearly 20 percent while we sleep, creating room for more growth and learning the next day.

Continue reading “How the Brain Resets During Sleep” | >

Way cool.

Feb. 2 (UPI) — Scientists at the University of California, Santa Barbara want to study the effects of various mechanical forces on individual brain cells. Until now, however, researchers didn’t have the right tools.

To study brain impacts at the nanoscale, researchers built the world’s tiniest hammer — the μHammer, or “microHammer.” The μHammer is a cellular-scale machine capable of applying a variety of mechanical forces to neural progenitor cells, brain-centric stem cells. Eventually, scientists hope to use the hammer to apply forces to neurons and neural tissue.

The hammer piggybacks on existing cell-sorting technology which isolates individual cells for diagnostics and immunotherapy. Once isolated, the machine can apply a range of forces. Post-impact structural and biomechanical analysis will allow scientists study the effects of focus in near real-time.

Continue reading “Scientists build world’s tiniest hammer to bang on brain cells” | >

A research team led by Professor Ed X. Wu of the Department of Electrical and Electronic Engineering at the University of Hong Kong has used an innovative neuroimaging tool to interrogate the complex brain networks and functions.

The team has successfully manipulated two pioneering technologies: optogenetics and imaging (fMRI), for investigation of the dynamics underlying activity propagation. Their breakthrough to simultaneously capture large-scale brain-wide neural activity propagation and interaction dynamics, while examining their functional roles has taken scientists a step further in unravelling the mysteries of the brain. It could lead to the development of new neurotechnologies for early diagnosis and intervention of brain diseases including autism, Alzheimer’s disease or dementia.

The findings have recently been published in the prestigious international academic journal Proceedings of the National Academy of Sciences (PNAS).

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Interesting read especially as we look at various areas including synbio and super humans.

The results are significant for gene therapy procedures and for our understanding of cell transformation. A team of researchers from the biology department at TU Darmstadt has discovered that the processes for repairing DNA damage are far more complex than previously assumed. The ends of breaks in the double helix are not just joined, they are first changed in a meticulously choreographed process so that the original genetic information can be restored. The results have now been published in the research journal Molecular Cell.

DNA, the carrier of our genetic information, is exposed to continual damage. In the most serious damage of all, the DNA double-strand break, both strands of the double helix are broken and the helix is divided in two. If breaks like this are not efficiently repaired by the cell, important genetic information is lost. This is often accompanied by the death of the cell, or leads to permanent genetic changes and cell transformation. Over the course of evolution, ways to repair this DNA damage have developed, in which many enzymes work together to restore the genetic information with the maximum possible precision.

As it stands today, there are two main ways of repairing DNA double-strand breaks, which differ greatly in terms of their precision and complexity. The apparently simpler method, so-called non-homologous end joining, joins together the break ends as quickly as possible, without placing particular importance on accurately restoring the damaged genetic information. The second method of repair, homologous recombination, on the other hand, uses the exactly identical information present on a sister copy to repair the damaged DNA with great precision. However, such sister copies are only present in dividing cells, as the genetic information has to be duplicated before the cells divide. But most cells in the human body do not undergo division, which therefore assigns them to the apparently more inaccurate method of end joining.

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To generate swarms of new viral particles, a virus hijacks a cell into producing masses of self-assembling cages that are then loaded with the genetic blueprint for the next infection. But the picture of how that DNA is loaded into those viral cages, or capsids, was blurry, especially for two of the most common types of DNA virus on earth, bacterial viruses and human herpesvirus. Jefferson researchers pieced together the three-dimensional atomic structure of a doughnut-shaped protein that acts like a door or ‘portal’ for the DNA to get in and out of the capsid, and have now discovered that this protein begins to transform its structure when it comes into contact with DNA. Their work published in Nature Communications.

“Researchers thought that the portal protein acts as an inert passageway for DNA,” says senior author Gino Cingolani, Ph.D., a Professor in the Department of Biochemistry and Molecular Biology at Thomas Jefferson University and researcher at the Sidney Kimmel Cancer Center. “We have shown that the portal is much more like a sensor that essentially helps measure out an appropriate length of DNA for each capsid particle, ensuring faithful production of new viral particles.”

The finding solves a longstanding puzzle in the field, and reveals a potential drug target for one of the most common human viral pathogens, herpesviruses, which is responsible for diseases such as chicken pox, mononucleosis, lymphomas and Kaposi sarcoma.

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