The technology I want to talk about today is something out of this world, but also a bit controversial There is a startup in Australia who are actually growing live human neurons and then integrating it into traditional computer chips⊠mind-blowing stuff!
Cerebral organoids at the air-liquid interface generate nerve tracts with functional output. https://www2.mrc-lmb.cam.ac.uk/cerebral-organoids-at-the-airâŠal-output/ âThe capacity for this model to be used to investigate the way in which neurons connect up within the brain and with the spinal cord could have important implications for our understanding of a range of diseases. In particular defects in neuronal connectivity are thought to underlie various psychiatric illnesses, including schizophrenia, autism, and depression. â
Cerebral organoids at the airâliquid interface generate diverse nerve tracts with functional output. https://www.readcube.com/articles/10.1038/s41593-019-0350-2 âFinally, through electrophysiological and co-culture studies, we demonstrate functionality of these tracts, which are even capable of eliciting coordinated muscle contractions in co-cultured mouse spinal cordâmuscle explants. This approach is likely to be a useful new tool, not only because of its ease, but also due to its util-ity in studying axon guidance, tract formation, and connectivity in a human systemâ
Whatâs Wrong With Growing Blobs of Brain Tissue? https://www.theatlantic.com/science/archive/2018/04/what-hapâŠns/558881/ âThe stuff we really care about in the brain, like consciousness, are emergent phenomenaâthey arise from the collective workings of individual neurons, which create a whole thatâs greater than the sum of its parts. The problem is that we donât know at what level these phenomena emerge. A neuron is not conscious. A person is. What about all the steps in the middle? What about 2 million neurons? 20 million? 200 million?â
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Researchers at Texas A&M University have developed the first molecular therapeutic for Angelman syndrome to advance into clinical development.
In a new article, published today in Science Translational Medicine, Dr. Scott Dindot, an associate professor and EDGES Fellow in the Texas A&M School of Veterinary Medicine and Biomedical Sciencesâ (VMBS) Department of Veterinary Pathobiology, and his team share the process through which they developed this novel therapeutic candidate, also known as 4.4.PS.L, or GTX-102. Dindot is also the executive director of molecular genetics at Ultragenyx, which is leading the development of GTX-102.
Angelman syndrome (AS) is a devastating, rare neurogenetic disorder that affects approximately 1 in 15,000 live births per year; the disorder is triggered by a loss of function of the maternal UBE3A gene in the brain, causing developmental delay, absent speech, movement or balance disorder, and seizures.
Changes in the brain caused by Alzheimerâs disease are associated with shortening of the telomeresâthe protective caps on the ends of chromosomes that shorten as cells ageâaccording to a new study led by Anya Topiwala of Oxford Population Health, part of the University of Oxford, UK, published March 22 in the open-access journal PLOS ONE.
Telomeres on chromosomes protect DNA from degrading, but every time a cell divides, the telomeres lose some of their length. Short telomeres are a sign of stress and cellular aging, and are also associated with a higher risk of neurological and psychiatric disorders. Currently, little is known about the links between telomere length and changes that occur in the brains of people with neurological conditions. Understanding those relationships could offer insights into the biological mechanisms that cause neurodegenerative disorders.
In the new study, researchers compared telomere length in white blood cells to results from brain MRIs and electronic health records from more than 31,000 participants in the UK Biobank, a large-scale biomedical database and research resource containing anonymized genetic, lifestyle and health information from half a million UK participants.
Watch as primordial neural cells dance across, grow into, and even move 3D scaffolds engineered to heal brain injury from stroke and other trauma. Decorating the scaffold with various nutrients and biochemical signals allow researchers to control what types of brain tissues they become.
âThis interface could revolutionize the way we interact with technology.â
Researchers from the University of Cambridge have created a new type of neural implant that could restore limb function in paralyzed limbs.
There have been former attempts at using neural implants to restore limb function, but these mostly failed. This is because scar tissue can envelop the electrodes over time, disrupting the connection between the device and the nerve.
University of Cambridge.
The developed device works in sync between the brain and paralyzed limbs â it combines flexible electronics and human stem cells to âbetter integrateâ with the nerve and drive limb function, according to a press release.
What if you could control a device, not with your hand, but with your mind? Physician and entrepreneur Tom Oxley talks about the implantable brain-computer interface that can change the way we think.
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