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Category: neuroscience – Page 102

Tiny plastic particles may accumulate at higher levels in the human brain than in the kidney and liver, with greater concentrations detected in postmortem samples from 2024 than in those from 2016, suggests a paper published in Nature Medicine. Although the potential implications for human health remain unclear, these findings may highlight a consequence of rising global concentrations of environmental plastics.

The amount of environmental nano-and microparticles, which range in size from as small as 1 nanometer (one billionth of a meter) up to 500 micrometers (one millionth of a meter) in diameter, has increased exponentially over the past 50 years. However, whether they are harmful or toxic to humans is unclear. Most previous studies used visual microscopic spectroscopy methods to identify particulates in , but this is often limited to particulates larger than 5 micrometers.

Researcher Matthew Campen and colleagues used novel methods to analyze the distribution of micro-and nanoparticles in samples of , kidney, and tissues from human bodies that underwent autopsy in 2016 and 2024. A total of 52 brain specimens (28 in 2016 and 24 in 2024) were analyzed.

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Attention-deficit hyperactivity disorder (ADHD) is a well-known neurodevelopmental disorder that affects the brain’s ability to regulate attention and control impulses. It poses many challenges to those affected, typically making it difficult for them to sustain focus, follow through with instructions, and maintain a calm and restful state.

As one of the most common neurodevelopmental disorders, ADHD impacts individuals throughout their lives, creating a breadth of social, emotional, academic, and workplace challenges.

Despite its high prevalence and decades of research, currently available drugs for ADHD are not able to completely resolve the core symptoms of the disorder in most cases.

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Children with attention deficit hyperactivity disorder (ADHD) do not have a behavioral disorder, nor are they lazy, or lacking in manners and boundaries. Their brains mature in a different way, with different patterns of neurological activity and a number of neurochemical differences. For this reason, ADHD is considered to be a neurodevelopmental disorder.

These neurological imbalances manifest as attention difficulties, disorganization, or hyperactivity and impulsivity. While these are most noticeable in childhood, where prevalence is estimated at 5%, ADHD can persist into adulthood, where prevalence is 2.5% of the population. ADHD can therefore have social, academic and occupational impacts throughout a person’s life.

Although there are risk factors (such as mothers smoking during pregnancy or ), these have not been shown to directly cause ADHD. Genetic factors play a more significant role, as 74% of cases are hereditary.

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I take Adderall.

Attention Deficit/Hyperactivity Disorder (ADHD) is one of the most prevalent neurodevelopmental disorders and can persist into adulthood in the majority of cases. ADHD is associated with deficits in cognitive functions, in particular executive functions such as motor and interference inhibition, sustained attention, working memory, timing, psychomotor speed, reaction time variability and switching.

This is the first meta-analysis paper of chronic medication effects on cognition in ADHD, looking at attention, inhibition, reaction time and working memory. All of these aspects can affect academic performance in school, and occupational performance in adults.

The research is published in the journal Neuroscience & Biobehavioral Reviews.

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Summry: New research reveals that dopamine plays a crucial role in teaching young male mice to fight, with the chemical’s influence diminishing as they gain experience. In novice fighters, boosting dopamine increased aggression, while blocking it stopped them from fighting.

However, experienced fighters showed no changes in behavior regardless of dopamine manipulation, highlighting the role of experience in shaping aggression. The study identifies the lateral septum as a key brain region for “aggression learning” in males, but no similar effect was observed in females.

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Recent research demonstrates that brain organoids can indeed “learn” and perform tasks, thanks to AI-driven training techniques inspired by neuroscience and machine learning. AI technologies are essential here, as they decode complex neural data from the organoids, allowing scientists to observe how they adjust their cellular networks in response to stimuli. These AI algorithms also control the feedback signals, creating a biofeedback loop that allows the organoids to adapt and even demonstrate short-term memory (Bai et al. 2024).

One technique central to AI-integrated organoid computing is reservoir computing, a model traditionally used in silicon-based computing. In an open-loop setup, AI algorithms interact with organoids as they serve as the “reservoir,” for processing input signals and dynamically adjusting their responses. By interpreting these responses, researchers can classify, predict, and understand how organoids adapt to specific inputs, suggesting the potential for simple computational processing within a biological substrate (Kagan et al. 2023; Aaser et al. n.d.).

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As people age, their memory and thinking skills naturally decline. Approximately 15% of older adults experience mild cognitive impairment, a major risk factor for dementia and other forms of dementia such as Alzheimer’s disease.

Since cognitive decline and dementia are growing public health concerns, scientists are working to better understand the risk factors and find ways to reduce them. One emerging area of research suggests that oral health may play a role in brain health.

Now, a new study suggests that the bacteria living in the mouth may influence cognitive function as people age, with some harmful bacteria possibly contributing to the development of dementia and Alzheimer’s disease.

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Light therapy is emerging as a promising, non-pharmacological treatment for Alzheimer’s.

Alzheimer’s disease is a progressive neurological disorder that primarily affects older adults, leading to memory loss, cognitive decline, and behavioral changes. It is the most common cause of dementia. The disease is characterized by the buildup of amyloid plaques and tau tangles in the brain, which disrupt cell function and communication. There is currently no cure, and treatments focus on managing symptoms and improving quality of life.

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