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Can we live long enough to live forever? Can we discover Immortality? Dr. Aubrey De Grey

Dr. Aubrey De Grey, SENS Foundation, Co-Founder, talks of species that live hundreds of years. We have to understand metabolism and postpone old age.

How can reverse aging by improving repair mechanism. Cells die, and the waste as well as energy byproducts to avoid tissue breakdown with methyl donors.

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Cellular senescence is one phenomenon by which normal cells cease to divide. In their seminal experiments from the early 1960’s, Leonard Hayflick.

Senescent cells increase in many tissues with aging; they also occur in organs associated with many chronic diseases and after radiation or chemotherapy. Senolytics are a class of drugs that selectively eliminate senescent cells.

Cancer cells are immortal, why?

Reversing Aging Update

Celebrate Unlimited Life Spans and a presentation by Bill Faloon on Human Age Reversal.
Bill Faloon will give an update on Age Reversal, similar to a presentation that he shared at RAAD Fest 2018!

Bill Faloon compiled the 1,500-page medical reference book Disease Prevention and Treatment, and his latest book is Pharmocracy: How Corrupt Deals and Misguided Medical Regulations Are Bankrupting America—and What to Do About It. He is also Director and Co-founder of the Life Extension Foundation.

To promote Life Extension’s innovative medical concepts, Bill has been featured in hundreds of media appearances including The Phil Donahue Show, The Joan Rivers Show, Tony Brown’s Journal, ABC News Day One, and Newsweek magazine.

Church of Perpetual Life, a science-based church is open to people of all faiths & belief systems. We are non-denominational & non-judgmental and a central gathering place of Transhumans. What unites us is our common faith, belief, and desire in Unlimited Life Spans.

New Weapon Against Gruesome Venomous Snakebites Is Invisible to the Eye

When it comes to venomous snake bites, time is tissue. Even non-fatal snake bites still rapidly kill skin and muscle in a gruesome process called necrosis, often leaving victims permanently disfigured. In an effort to help reduce the global health burden of these bites, a team of scientists has developed an antivenom cocktail that saves tissue after a snake bite, sparing survivors a lifetime of disability.

In a paper published Thursday in the journal PLOS Neglected Tropical Diseases, researchers demonstrate that their formula, when injected into mice that had been exposed to venom from a black-necked spitting cobra (Naja nigricollis), protected against any tissue-killing effects. What’s unique about their new treatment is that it’s not made up of any one substance but a mixture of nanoparticles, which can target the individual compounds that make up a snake’s poison.

“If this is achieved, then the progression of this local necrosis would be halted, and then the person can be transported to a health facility to receive the antivenom, but the local tissue damage would have been controlled and the frequency of permanent tissue damage and sequelae would be reduced,” José María Gutiérrez, Ph.D.. a senior professor of microbiology at Instituto Clodomiro Picado (the University of Costa Rica) and one of the paper’s authors, tells Inverse.

Physicist Who Coined the ‘God Particle’ and Sold His Nobel Prize to Pay Medical Bills Dies at 96

Leon Lederman, the former head of the Fermi National Accelerator Lab and winner of the Nobel Prize in physics in 1988, died at a nursing home in Idaho on October 3rd. He was 96.

Lederman will perhaps best be remembered for coining the phrase “the God particle,” referring to the Higgs boson, which was theorized for decades before it was finally observed in 2012.

Scarlet Protein Might Protect Against Parkinson’s Disease

Scarlet protein has a protective effect against Parkinson’s disease in fruit flies.


Researchers at the Department of Biological Sciences at Lehigh University in Bethlehem, Pennsylvania, discovered that a protein known as Scarlet has protective effects against the fruit fly version of Parkinson’s disease [1].

Abstract

Parkinson’s Disease (PD) is characterized by the loss of dopaminergic neurons, resulting in progressive locomotor dysfunction. Identification of genes required for the maintenance of these neurons should help to identify potential therapeutic targets. However, little is known regarding the factors that render dopaminergic neurons selectively vulnerable to PD. Here we show that Drosophila melanogaster scarlet mutants exhibit an age-dependent progressive loss of dopaminergic neurons, along with subsequent locomotor defects and a shortened lifespan. Knockdown of Scarlet specifically within dopaminergic neurons is sufficient to produce this neurodegeneration, demonstrating a unique role for Scarlet beyond its well-characterized role in eye pigmentation.