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Category: biotech/medical – Page 234

A new photonic processor efficiently solves complex NP-complete problems using light, offering faster computation and scalability for future applications in optical neural networks and quantum computing.

As technology continues to evolve, the limitations of traditional electronic computers are becoming more evident, particularly when addressing highly complex computational problems. NP-complete problems, which grow exponentially in difficulty as their size increases, are among the most challenging in computer science. These issues affect a wide range of fields, from biomedicine to transportation and manufacturing. To find more efficient solutions, researchers are turning to alternative computing methods, with optical computing showing significant promise.

Breakthrough in Photonic Processor Development.

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Michael Levin is a Distinguished Professor in the Biology department at Tufts University and associate faculty at the Wyss Institute for Bioinspired Engineering at Harvard University. @drmichaellevin holds the Vannevar Bush endowed Chair and serves as director of the Allen Discovery Center at Tufts and the Tufts Center for Regenerative and Developmental Biology. Prior to college, Michael Levin worked as a software engineer and independent contractor in the field of scientific computing. He attended Tufts University, interested in artificial intelligence and unconventional computation. To explore the algorithms by which the biological world implemented complex adaptive behavior, he got dual B.S. degrees, in CS and in Biology and then received a PhD from Harvard University. He did post-doctoral training at Harvard Medical School, where he began to uncover a new bioelectric language by which cells coordinate their activity during embryogenesis. His independent laboratory develops new molecular-genetic and conceptual tools to probe large-scale information processing in regeneration, embryogenesis, and cancer suppression.

TIMESTAMPS:
0:00 — Introduction.
1:41 — Creating High-level General Intelligences.
7:00 — Ethical implications of Diverse Intelligence beyond AI & LLMs.
10:30 — Solving the Fundamental Paradox that faces all Species.
15:00 — Evolution creates Problem Solving Agents & the Self is a Dynamical Construct.
23:00 — Mike on Stephen Grossberg.
26:20 — A Formal Definition of Diverse Intelligence (DI)
30:50 — Intimate relationships with AI? Importance of Cognitive Light Cones.
38:00 — Cyborgs, hybrids, chimeras, & a new concept called “Synthbiosis“
45:51 — Importance of the symbiotic relationship between Science & Philosophy.
53:00 — The Space of Possible Minds.
58:30 — Is Mike Playing God?
1:02:45 — A path forward: through the ethics filter for civilization.
1:09:00 — Mike on Daniel Dennett (RIP)
1:14:02 — An Ethical Synthbiosis that goes beyond “are you real or faking it“
1:25:47 — Conclusion.

EPISODE LINKS:
- Mike’s Round 1: https://youtu.be/v6gp-ORTBlU
- Mike’s Round 2: https://youtu.be/kMxTS7eKkNM
- Mike’s Channel: https://www.youtube.com/@drmichaellevin.
- Mike’s Website: https://drmichaellevin.org/
- Blog Website: https://thoughtforms.life.
- Mike’s Twitter: https://twitter.com/drmichaellevin.
- Mike’s Publications: https://scholar.google.com/citations?user=luouyakAAAAJ&hl=en.
- Mike’s NOEMA piece: https://www.noemamag.com/ai-could-be-a-bridge-toward-diverse-intelligence/
- Stephen Grossberg: https://youtu.be/bcV1eSgByzg.
- Mark Solms: https://youtu.be/rkbeaxjAZm4
- VPRO Roundtable: https://youtu.be/RVrnn7QW6Jg?feature=shared.

CONNECT:
- Website: https://tevinnaidu.com.
- Podcast: https://podcasters.spotify.com/pod/show/drtevinnaidu.
- Twitter: https://twitter.com/drtevinnaidu.
- Facebook: https://www.facebook.com/drtevinnaidu.
- Instagram: https://www.instagram.com/drtevinnaidu.
- LinkedIn: https://www.linkedin.com/in/drtevinnaidu.

Disclaimer: The information provided on this channel is for educational purposes only. The content is shared in the spirit of open discourse and does not constitute, nor does it substitute, professional or medical advice. We do not accept any liability for any loss or damage incurred from you acting or not acting as a result of listening/watching any of our contents. You acknowledge that you use the information provided at your own risk. Listeners/viewers are advised to conduct their own research and consult with their own experts in the respective fields.

#MichaelLevin #DiverseIntelligence #AI #Mind

Continue reading “Michael Levin: What is Synthbiosis? Diverse Intelligence Beyond AI & The Space of Possible Minds” | >

In a laboratory set-up simulating the human stomach and intestine, researchers at the University of Amsterdam have explored the fate of plastic nanoparticles during gastrointestinal digestion. In their paper published in the October issue of Chemosphere, they report how a range of model plastic nanoparticles interact with digestive enzymes and form agglomerates.

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The future of therapeutic apheresis & transfusion medicine — dr. tina ipe, MD, MPH — CEO, regen med clinic.

Dr. Tina Ipe, MD, MPH is Chief Executive Officer at Regen Med Clinic (https://www.regenmed.vip/), a medical practice which provides multi-specialty infusions, cutting-edge treatments such as therapeutic apheresis (plasmapheresis and collections), as well as novel aesthetic treatments, for patients with a variety chronic illnesses.

Dr. Ipe is a board-certified physician and clinical researcher. Before entering private practice, she was Chief Medical Officer at the Oklahoma Blood Institute, Associate Medical Director at Houston Methodist Hospital, and Division Director at University of Arkansas for Medical Sciences (UAMS). She is an expert in the fields of blood disorders, immunology, therapeutic apheresis, blood banking, and transfusion medicine. She has published more than 50 peer-reviewed manuscripts and book chapters.

Dr. Ipe received her medical degree from the Medical College of Virginia and completed her residency in clinical pathology and fellowship in transfusion medicine at the Hospital of the University of Pennsylvania. She also has an MPH from Dartmouth.

#TinaIpe #BloodDisorders #Blood #Biotherapies #Immunology #TherapeuticApheresis #BloodBanking #TransfusionMedicine #RegenerativeMedicine #TherapeuticPlasmaExchange #ConvalescentPlasma #ExtracorporealPhotopheresis #Parabiosis #Longevity #Aging #HospiceCare #STEM #Innovation #Science #Technology #Research #ProgressPotentialAndPossibilities #IraPastor #Podcast #Podcaster #Podcasting #ViralPodcast

Continue reading “Dr Tina Ipe, MD — CEO, Regen Med Clinic — The Future Of Therapeutic Apheresis & Transfusion Medicine” | >

Actin, a family of proteins that help give cells their shape, are abundant throughout the body.

Humans aren’t the only ones who grow forgetful as they age—fruit flies do, too. But because fruit flies have a lifespan of only about two months, they can be a useful model for understanding the cognitive decline that comes with aging.

A new study published in Nature Communications shows that when a common cell structural protein called filamentous actin, or F-actin, builds up in the brain, it inhibits a key process that removes unnecessary or dysfunctional components within cells, including DNA, lipids, proteins and organelles.

The resulting accumulation of waste diminishes neuronal functions and contributes to . By tweaking a few in aging fruit flies’ neurons, the researchers prevented F-actin buildup, maintained cellular recycling and extended the healthy lifespan of fruit flies by approximately 30%.

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There are rare cells in the gut called enteroendocrine cells (EECs) that could be manipulated in a variety of ways to detect or treat disease.

The trillions of microbes in our gastrointestinal tract, known as the gut microbiome, are crucial to the body; the gut microbiome aids in digestion, nutrient absorption, and influences our health in different ways. But the body also has to be protected from all of those microbes, which are kept behind a tight barrier. But if the intestinal barrier is dysfunctional, or leaky, serious problems can arise.

There are cells in the gut called enteroendocrine cells (EECs) that can generate hormones, which may have a variety of effects on the body. EECs release hormones in response to cues like food intake and stomach stretching. The hormones can then influence physiological processes related to digestion or appetite. Scientists have now found receptors on EECs that control hormone release. It may one day be possible to alter these receptors to treat disease. The research has been reported in Science.

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Organization runs deep in our family tree, if we use the literal definition of “organize”: to be furnished with organs. Eukaryotes emerged billions of years ago, bringing with them the copious benefits of compartmentalization.

All modern multicellular life — all life that any of us regularly see — is made of cells with a knack for compartmentalization. Recent discoveries are revealing how the first eukaryote got its start.

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One prevailing hypothesis is that physical fitness mitigates structural brain changes that contribute to cognitive decline. Recent evidence points to a potential role involving myelin —the insulating sheath surrounding neurons that is crucial for efficient neural signaling and overall cognitive health. Myelination facilitates rapid signal transmission and supports neural network integrity.

The degeneration of myelin in the brain is increasingly recognized as a critical factor contributing to disruptions in neural communication, which may play a significant role in the cognitive decline observed in Alzheimer’s disease and other neurodegenerative disorders. Emerging research suggests that myelin breakdown may even precede the formation of amyloid-beta plaques and neurofibrillary tangles—the hallmark pathological features of Alzheimer’s disease. Advanced imaging studies have detected early myelin degeneration in individuals who later develop Alzheimer’s, indicating that myelin damage could be an initial event in the disease’s progression.

Age-related deterioration of myelin is closely associated with cognitive decline. Reduced white matter integrity—often resulting from myelin damage—is correlated with declines in memory, executive function, and processing speed in older adults. As myelin degradation leads to the slowing of cognitive processes and disrupts the synchronization of neural networks, preserving myelin integrity is essential for sustaining cognitive health across the lifespan.

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What keeps some immune systems youthful and effective in warding off age-related diseases? In a new paper published in Cellular & Molecular Immunology, USC Stem Cell scientist Rong Lu and her collaborators point the finger at a small subset of blood stem cells, which make an outsized contribution to maintaining either a youthful balance or an age-related imbalance of the two main types of immune cells: innate and adaptive.

Innate immune cells serve as the body’s first line of defense, mobilizing a quick and general attack against invading germs. For germs that evade the body’s innate immune defenses, the second line of attack consists of , such as B cells and T cells that rely on their memory of past infections to craft a specific and targeted response. A healthy balance between innate and adaptive immune cells is the hallmark of a youthful immune system—and a key to longevity.

“Our study provides compelling evidence that when a small subset of overproduces innate immune cells, this drives the aging of the immune system, contributes to disease, and ultimately shortens the lifespan,” said Lu, who is an associate professor of stem cell biology and , , medicine, and gerontology at USC, and a Leukemia & Lymphoma Society Scholar. Lu is also a member of the Eli and Edythe Broad Center for Regenerative Medicine and Stem Cell Research at USC, and the USC Norris Comprehensive Cancer Center at the Keck School of Medicine of USC.

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