A team of NYU chemists and physicists are using cutting-edge tools—holographic microscopy and super-resolution imaging—to unlock how cells build and grow tiny, dynamic droplets known as biomolecular condensates.

IN A NUTSHELL 🐕 CARA is a robot dog created by a Purdue University student using innovative capstan drive technology. 🔧 The robot incorporates custom 3D-printed parts and high-strength materials like carbon fiber for durability and efficiency. 🤖 Advanced coding techniques such as Inverse Kinematics allow CARA to move with natural grace and agility. 🚀
Cytonics: Our mission is to rid the world of the pain, suffering, and debilitated quality of life caused by Osteoarthritis (OA).
Cytonics: Our mission is to rid the world of the pain, suffering, and debilitated quality of life caused by Osteoarthritis (OA).
The skeletal muscles of men and women process glucose and fats in different ways. A study conducted by the University Hospital of Tübingen, the Institute for Diabetes Research and Metabolic Diseases of Helmholtz Munich and the German Center for Diabetes Research (DZD) e. V. provides the first comprehensive molecular analysis of these differences. The results, published in Molecular Metabolism, possibly give an explanation for why metabolic diseases such as diabetes manifest differently in women and men—and why they respond differently to physical activity.
Skeletal muscles are far more than just “movement driving motors.” They play a central role in glucose metabolism and therefore also in the development of type 2 diabetes. This is due to the fact that around 85% of insulin-dependent glucose uptake takes place in the muscles.
This means that if muscle cells react less sensitively to insulin, for example in the case of insulin resistance, glucose is less easily absorbed from the blood. This process is specifically counteracted by physical activity.
Using data and digitalization technologies could revolutionize industries and contribute to net-zero climate goals.
Questions to inspire discussion.
🤝 Q: What are the potential issues with the Uber-Lucid-Neuro robotaxi partnership? A: The partnership is a “cluster f waiting to happen” due to independent entities involved, which typically end in a “messy divorce”, making it potentially uncompetitive against fully integrated solutions like Tesla’s.
🗺️ Q: How does Tesla’s robotaxi service area expansion compare to Waymo’s? A: Tesla expanded its service area in 22 days, while Waymo’s first service area expansion in Austin, Texas took 4 months and 13 days, demonstrating Tesla’s faster and more aggressive approach to expansion.
Business Viability.
💼 Q: What concerns exist about the Uber-Lucid-Neuro robotaxi partnership’s business case? A: While considered a “breakout moment” for autonomous vehicles, the business case and return on investment for the service remain unclear, according to former Ford CEO Mark Fields.
🏭 Q: What manufacturing advantage does Tesla have in the robotaxi market? A: Tesla’s fully vertically integrated approach and ability to mass-manufacture Cyber Cabs at a scale of tens of thousands per month gives it a significant cost-per-mile advantage over competitors using more expensive, non-specialized vehicles. ## Key Insights.
In a major advance for patients with Crohn’s disease, a new study led by researchers at Mount Sinai Health System found that guselkumab, a medication with a mechanism of action that is new to inflammatory bowel disease (IBD) treatment, outperformed an established standard of care in promoting intestinal healing and symptom relief.
These findings from two pivotal Phase III trials known as GALAXI 2 and 3, published in The Lancet, provided the basis for the recent Food and Drug Administration approval of guselkumab (brand name Tremfya) for the treatment of moderately to severely active Crohn’s disease.
Crohn’s disease affects roughly 780,000 people in the United States and often requires a lifetime of management. Despite numerous available biologic medications, many patients fail to achieve sustained remission. Guselkumab blocks the interleukin-23 (IL-23) pathway, a key driver of chronic intestinal inflammation.