The ancient evolution of fish mouths could help solve a modern source of plastic pollution.
Inspired by these natural filtration systems, scientists in Germany have invented a way to remove 99 percent of plastic particles from water. It’s based on how some fish filter-feed to eat microscopic prey.
The research team has already filed a patent in Germany, and in the future, they hope their creation will help curb a ubiquitous form of plastic pollution that many are unaware of.
Transposable elements in cancer therapy.
Transposable elements (TEs) are a major source of immunogenic nucleic acids that can be therapeutically reactivated in cancer cells to induce a state of viral mimicry.
TE expression can trigger innate immune sensing pathways, including type I interferon responses, and promote immunogenic cell death via sensors such as RIGI, MDA5, cGAS, and Z-DNA binding protein 1.
Although initially described in the context of epigenetic therapies, viral mimicry is now recognized as a shared response to diverse cancer treatment modalities, including chemotherapies and targeted therapies.
Despite their distinct primary mechanisms, these treatments converge on TE reactivation through disruption of DNA/histone methylation, p53 activation, and perturbation of mRNA splicing.
Therapeutic resistance to chemotherapy, radiation, and targeted agents is associated with TE silencing, identifying TE repression as a targetable axis of resistance.
Combination strategies to induce immunogenic TE expression can further enhance viral mimicry and boost antitumor immunity. https://sciencemission.com/Viral-mimicry-in-cancer-therapy
How common are Earth-like planets in the universe? When I started working on supernova explosions, I never imagined that my research would eventually lead me to ask a question about the origin of Earth-like planets. Yet that is exactly where it brought me.
For decades, planetary scientists have believed that the early solar system was enriched with short-lived radioactive elements—such as aluminum-26—by a nearby supernova. These radioactive elements played a crucial role in forming water-depleted rocky planets such as Earth. Their decay heated young planetesimals, causing them to lose much of their originally accreted water and other volatile materials.
There was just one problem that kept bothering me.
One day they may come for us.
Are Berserker probes hunting advanced life? Exploring the deadliest Fermi Paradox solution.
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Scientists studying Alzheimer’s in African Americans have uncovered a striking genetic clue that may cut across racial lines. In brain tissue from more than 200 donors, the gene ADAMTS2 was significantly more active in people with Alzheimer’s than in those without it. Even more surprising, this same gene topped the list in an independent study of White individuals. The discovery hints at a common biological pathway behind Alzheimer’s and opens the door to new treatment strategies.
🧠 VIDEO SUMMARY:
CRISPR gene editing in 2025 is no longer science fiction. From curing rare immune disorders and type 1 diabetes to lowering cholesterol and reversing blindness in mice, breakthroughs are transforming medicine today. With AI accelerating precision tools like base editing and prime editing, CRISPR not only cures diseases but also promises longer, healthier lives and maybe even longevity escape velocity.
0:00 – INTRO — First human treated with prime editing.
0:35 — The DNA Problem.
1:44 – CRISPR 1.0 — The Breakthrough.
3:19 – AI + CRISPR 2.0 & 3.0
4:47 – Epigenetic Reprogramming.
5:54 – From the Lab to the Body.
7:28 – Risks, Ethics & Power.
8:59 – The 2030 Vision.
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Part 2 – “Longevity Escape Velocity 2025: Latest Research Uncovered!“
Part 3 – “Longevity Escape Velocity: How AI is making us immortal by 2030!”
📌 Easy Insight simplifies the future — from longevity breakthroughs to mind-bending AI and quantum revolutions.
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Scodellaro, R., Kulkarni, A., Alves, F. et al. Training convolutional neural networks with the Forward–Forward Algorithm. Sci Rep 15, 38,461 (2025). https://doi.org/10.1038/s41598-025-26235-2
A new study reveals that bacteria can survive antibiotic treatment through two fundamentally different “shutdown modes,” not just the classic idea of dormancy. The paper is published in the journal Science Advances.
The researchers show that some cells enter a regulated, protective growth arrest, a controlled dormant state that shields them from antibiotics, while others survive in a disrupted, dysregulated growth arrest, a malfunctioning state marked by vulnerabilities, especially impaired cell membrane stability. This distinction is important because antibiotic persistence is a major cause of treatment failure and relapsing infections even when bacteria are not genetically resistant, and it has remained scientifically confusing for years, with studies reporting conflicting results.
By demonstrating that persistence can come from two distinct biological states, the work helps explain those contradictions and provides a practical path forward: different persister types may require different treatment strategies, making it possible to design more effective therapies that prevent infections from coming back.