Nvidia’s largest customers (e.g., Google, Amazon, Microsoft, Meta, and OpenAI) are developing AI chips that compete with Nvidia, presenting a potential long-term threat to the AI leader.
Nvidia could become a tech licensing powerhouse.
Imagine stepping into an airport where queues are relics of the past, replaced by seamless journeys orchestrated by intelligent machines. This isn’t science fiction – it’s the dawn of Airport 4.0, the cognitive era where airports transform from transit hubs into dynamic, personalized experiences.
As a frequent traveler myself, I’ve spent countless hours navigating the labyrinthine world of airports. The frustration of long lines, the stress of security checks, the wasted time waiting – it’s all too familiar. But Airport 4.0 paints a radically different picture. Facial recognition whisks me past security, AI-powered apps anticipate my needs, and personalized recommendations guide me to hidden gems within the terminal. This isn’t just a convenience; it’s a paradigm shift that unlocks a world of possibilities. Today, as we stand on the brink of the cognitive era, I’m keen to share my insights on how Airport 4.0 is reshaping the future of air travel, making it not just a journey from A to B but an experience in its own right.
A new report on Future of Airports from Markets and Markets Foresighting team delves into what will be a future airport.
Will AI Undermine Human Dignity?
Posted in robotics/AI
If AI can do parts of your job with greater efficiency and wisdom, how does that make you feel about your own self-worth?
The rise of AI will change how people consider their own self-worth and their ability to impact their communties for the better. Let’s see how this unfolds.
Scientists at the University of Pennsylvania have unveiled a revolutionary method to study the microscopic structures of the human brain. The study, led by Benjamin Creekmore in the labs of Yi-Wei Chang and Edward Lee, promises to enhance our understanding of various brain diseases, including Alzheimer’s and multiple sclerosis.
Cryo-electron tomography takes center stage
Traditionally, scientists have utilized electron microscopy to explore and comprehend the intricate details of cellular structures within the brain. However, this method has been fraught with challenges, such as the alteration of cell structures due to the addition of chemicals and physical tissue cutting.
Classical computers can sometimes outperform quantum computers thanks to new algorithms, challenging the idea that quantum always prevails.
NYU researchers have developed a new method that allows classical computers to perform certain tasks faster and more efficiently than quantum computers.
The new project will develop robotic satellite technologies to reliably connect with other satellites to perform in-orbit repairs and maintenance.
Explore the future of space technology as the University of Sydney pioneers a $2.3 million initiative in collaboration with industry leaders.
Altman estimates that he would need between $5 trillion and $7 trillion to overhaul the semiconductor industry, which is currently dominated by Nvidia, the leading provider of graphics processing units (GPUs) for AI applications. Nvidia’s market cap has soared to $1.72 trillion in 2023, surpassing many tech giants such as Amazon and Alphabet. Altman wants to challenge Nvidia’s monopoly and create more competition and innovation in the AI chip market.
White House’s $11 billion bet on US semiconductor
Meanwhile, The White House announced the US government’s plan to spend $11 billion on semiconductor-related research and development on Friday. This move comes in the wake of Congress approving the Chips and Science Act in August 2022, which provides $52.7 billion for semiconductor production and R&D. Of this, $39 billion is allocated for subsidies and $11 billion for R&D.
Fields medalist Cédric Villani explains some of John Nash’s most amazing theorems.
Fields medal winner Cédric Villani takes us through the very special world of mathematical creation of John Nash, who founded several new chapters of game theory and geometric analysis in just a few revolutionary contributions that seemed to come from nowhere.
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Buy Cedri’s book \.
Australian researchers have worked out how to fix a defect that causes lupus, and hope their world-first discovery will offer effective long-term treatment.
Published in Nature Communications, the Monash University-led study found a way to reprogram the defective cells of lupus patients with protective molecules from healthy people.
Using human cells, the new treatment restores the protective side of the immune system that prevents autoimmunity, which is when the immune system attacks its own cells. The findings relate to the autoimmune disease lupus, a debilitating disease with no cure and limited treatments.
Real-World Development of
Posted in biotech/medical, genetics
Patients with recurrent Escherichia coli bacteremia can harbor strains with mutations that promote multidrug antibiotic resistance:
Certain patients with gram-negative bacterial bloodstream infections (GNB-BSIs) are well-known to experience recurrent bacteremia after receiving antimicrobial therapy — but is this phenomenon due to microbial factors? Researchers have analyzed isolates from patients with relapsed GNB-BSIs in which the initial and subsequent strains were nearly identical genetically.
Paired bacteremic isolates of E. coli, Klebsiella species, Serratia marcescens, and Pseudomonas aeruginosa were identified for a detailed analysis of the E. coli strains. Time-kill studies found that 4 of the 11 recurrent isolates had a higher number of bacterial colony-forming units persisting through 24 hours of exposure to meropenem. The recurrent strain with the greatest number of persisting cells had a loss-of-function mutation in the ptsI gene (involved in the phosphoenolpyruvate phosphotransferase system and shown in vitro to be important to the effects of bactericidal antibiotics). Challenging mice with the initial and ptsI mutant recurrent strains in a bacteremia model showed that both variants were equally virulent, but the recurrent strain was 10-fold less susceptible to treatment with ertapenem.
This work affirms the clinical importance of persister bacterial strains in relapsing infections while also confirming the role of bacterial metabolic pathways in the development of antibiotic resistance. The results also raise the question of whether more-prolonged antibiotic therapy might be appropriate in individuals who develop recurrent E. coli bacteremia without an obvious host-related explanation.