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But then Santamaria, who is at the University of Calgary in Canada, came up with a bold idea. Maybe he could use these particles as a therapy to target and quiet, or even kill, the cells responsible for driving the disease — those that destroy insulin-producing islet cells in the pancreas. It seemed like a far-fetched idea, but he decided to try it. “I kept doing experiment after experiment,” he says. Now, more than two decades later, Santamaria’s therapy is on the cusp of being tested in people.
It’s not alone. Researchers have been trying for more than 50 years to tame the cells that are responsible for autoimmune disorders such as type 1 diabetes, lupus and multiple sclerosis. Most of the approved therapies for these conditions work by suppressing the entire immune response. This often alleviates symptoms but leaves people at elevated risk of infections and cancers.
But for decades, immunologists have hoped to restore what’s known as tolerance — the immune system’s ability to ignore antigens that belong in the body while appropriately attacking those that don’t. In some cases, that means administering the very antigens that the rogue cells are trained to attack, a strategy that can deprogram the cells and dampen the autoimmune response. Other researchers are trying to selectively wipe out the problematic cells, or to introduce suppressive immune cells that have been engineered to target them. One approach that relies on engineered immune cells was used to treat 15 people with lupus or other immune disorders with surprising success1. One participant has been symptom-free for more than two and a half years.
Artificial intelligence (AI) has become an indispensable component in the analysis of microscopic data. However, while AI models are becoming better and more complex, the computing power and associated energy consumption are also increasing.
Researchers at the Leibniz-Institut für Analytische Wissenschaften (ISAS) and Peking University have therefore created a free compression software that allows scientists to run existing bioimaging AI models faster and with significantly lower energy consumption.
The researchers have presented their user-friendly toolbox, called EfficientBioAI, in an article published in Nature Methods.
The infections typically occur in the upper Midwest and parts of the Southeast. A new study suggests the fungi’s range has expanded to Vermont.
It’s all thanks to what is called the “solar maximum,” when the sun is reaching its peak of a roughly 11-year cycle, which NASA says began again in December 2019. The sun’s activity has been ramping up since then, with an expected peak in July 2025.
MORE: Pilot performs mid-flight maneuver to give passengers a rare view of the northern lights
The northern lights, also known as the aurora borealis, happen in regions around the earth’s magnetic pole. They appear when electrons from solar flares interact with atoms and molecules in the Earth’s atmosphere. That in turn creates lights and multiple colors in the sky.
New angle for increasing longevity:
In a new study, scientists found that T cells can be tweaked to fight damaged cells, called senescent cells, associated with aging.
I hope more research is fruitful because I got the doctor’s results from my recent colonoscopy. The polyps are benign but pre cancerous. I go back in 5 years and I’ll be eating healthier and exercising. I gotta admit I don’t feel great but it could be something I ate.
Scientists in China have demonstrated a new kind of antidepressant could also have the potential to restore the body’s ability to fight some types of cancer.
In strategic combination with anti-tumor drugs, the oral antidepressant ansofaxine hydrochloride appears to inhibit colon cancer cell growth in cell cultures and in mice, strengthening the immune system and inducing a form of programmed cell death.
Continue reading “New Antidepressant Could Restore The Body’s Ability to Fight Cancer” »
Alibaba presents Ditto.
Large language models are superpositions of all characters: attaining arbitrary role-play via self-alignment.
Join the discussion on this paper page.
