Lifeboat Foundation

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Researchers from NYU discovered that classical computers could keep up with or even surpass quantum computers in certain circumstances. Classical computers can get a boost in speed and accuracy by adopting a new innovative algorithmic method, which could mean that they still have a future in a world of quantum computers.

Many experts believe that quantum computing is the future, and that we are veering away from classical computing, primarily because classical computers are significantly slower and weaker than their quantum-based counterparts. However, turns out that quantum computers are delicate and prone to information loss, and even if information is preserved it is difficult to convert it to classical information necessary for practical computation.

The indoor farming startup uses repurposed warehouse robots to grow produce for Amazon Fresh.

Compared to the Meta Quest 3, Meta CEO Mark Zuckerberg argues the Apple Vision Pro is packed with ‘trade-offs.’

A new approach has allowed researchers at Aalto University to create a kind of metamaterial that has so far been beyond the reach of existing technologies. Unlike natural materials, metamaterials and metasurfaces can be tailored to have specific electromagnetic properties, which means scientists can create materials with features desirable for industrial applications.

The new metamaterial takes advantage of the nonreciprocal magnetoelectric (NME) effect. The NME effect implies a link between specific properties of the material (its magnetization and polarization) and the different field components of light or other electromagnetic waves. The NME effect is negligible in natural materials, but scientists have been trying to enhance it using metamaterials and metasurfaces because of the technological potential this would unlock.

The work is published in the journal Nature Communications.

A new kind of implant could one day make it far easier for people with type 1 diabetes to manage their disease. The insulin-making implant is a mixture of transplanted islets cells and medical technology, inserted just below the skin in a person’s arm, and if it perform well in clinical trials, it could potentially last for years.

The challenge: Insulin is a hormone that our bodies use to convert sugar in our blood into energy. People with type 1 diabetes don’t produce enough (or any) insulin — if left untreated, this causes dangerously high blood sugar levels, leading to serious health issues or even death.

Regularly checking blood sugar levels and injecting synthetic insulin when they’re high is the most common way to treat type 1 diabetes, but it isn’t the only way.

Speaker: Dr. Zhongrui WangAbstract: The rapid development in the field of artificial intelligence has relied principally on the advances in computing hardwar…

Jesse Watters discusses the very vague alleged threat to Americans that lawmakers are keeping under wraps on ‘Jesse Watters Primetime.’

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Cecile G. Tamura ‎Lifeboat Foundation An effective treatment for depression from a systematic review of 200 unique RCTs:

Exercise.

Objective To identify the optimal dose and modality of exercise for treating major depressive disorder, compared with psychotherapy, antidepressants, and control conditions.

Continue reading “Effect of exercise for depression: systematic review and network meta-analysis of randomised controlled trials” »

“Focus has been on how cells become mechanosensitive, how cell adhesion became mechanosensitive through the recruitment of mechanosensitive proteins like talin, vinculin, and also other proteins,” said Sangyoon Han, a mechanobiologist at Michigan Technological University who was not involved in the study. “But not many studies were around to tell if there is any kind of premechanosensitive processes that act as a seed for the original binding of this mechanosensitive binding.”

Cooper’s team imaged adhesion formation at the cell surface interface using total internal reflection (TIRF) microscopy and observed that activated Cas clustered at this site almost a full minute before mechanosensing proteins arrived or β1 integrin was activated. They depleted Cas with siRNA and inactivated it to determine this protein’s role in the developing adhesion site. “When we remove Cas from cells, or inhibit its phosphorylation, or inhibit stuff downstream of Cas, the integrin clustering doesn’t really happen,” Cooper said. The Cas-depleted cells attached poorly to surfaces and were immobile.

Finally, the group identified a positive feedback loop between Cas and Rac1, which drives actin polymerization and is important in forming focal complexes that promoted cell adhesion formation.^5,6 Cas phosphorylation activated Rac1, which in turn generated reactive oxygen species, which promoted additional Cas activation. Cas degradation regulated this loop.