The infections typically occur in the upper Midwest and parts of the Southeast. A new study suggests the fungi’s range has expanded to Vermont.
It’s all thanks to what is called the “solar maximum,” when the sun is reaching its peak of a roughly 11-year cycle, which NASA says began again in December 2019. The sun’s activity has been ramping up since then, with an expected peak in July 2025.
MORE: Pilot performs mid-flight maneuver to give passengers a rare view of the northern lights
The northern lights, also known as the aurora borealis, happen in regions around the earth’s magnetic pole. They appear when electrons from solar flares interact with atoms and molecules in the Earth’s atmosphere. That in turn creates lights and multiple colors in the sky.
New angle for increasing longevity:
In a new study, scientists found that T cells can be tweaked to fight damaged cells, called senescent cells, associated with aging.
I hope more research is fruitful because I got the doctor’s results from my recent colonoscopy. The polyps are benign but pre cancerous. I go back in 5 years and I’ll be eating healthier and exercising. I gotta admit I don’t feel great but it could be something I ate.
Scientists in China have demonstrated a new kind of antidepressant could also have the potential to restore the body’s ability to fight some types of cancer.
In strategic combination with anti-tumor drugs, the oral antidepressant ansofaxine hydrochloride appears to inhibit colon cancer cell growth in cell cultures and in mice, strengthening the immune system and inducing a form of programmed cell death.
Continue reading “New Antidepressant Could Restore The Body’s Ability to Fight Cancer” »
Alibaba presents Ditto.
Large language models are superpositions of all characters: attaining arbitrary role-play via self-alignment.
Join the discussion on this paper page.
Researchers at City of Hope and Translational Genomics Research Institute (TGen) have developed and tested an innovative machine-learning approach that could one day enable the earlier detection of cancer in patients by using smaller blood draws. The study is published in the journal Science Translational Medicine.
“A huge body of evidence shows that cancer caught at later stages kills people. This new technology gets us closer to a world where people will receive a blood test annually to detect cancer earlier when it is more treatable and possibly curable,” said Cristian Tomasetti, Ph.D., corresponding author of the new study and director of City of Hope’s Center for Cancer Prevention and Early Detection.
Tomasetti explained that 99% of people diagnosed with Stage 1 breast cancer will be alive five years later; however, if it is found at Stage 4, when disease has spread to other organs, the five-year survival drops to 31%.
Aging is a common phenomenon among organisms, however, lifespan tends to vary across different species to a significant extent among vertebrates themselves. Aging occurs due to the gradual increase in DNA damage, disruption of cellular organelles, deregulation of protein function, disrupted metabolism and oxidative stress [1].
Longevity. Technology: The differences in lifespan are driven by trade-offs and evolutionary trajectories in the genomes of organisms. Age-specific selection also impacts allele (variations of a gene) frequencies in a population. This in turn impacts environment-specific mortality risk and disease susceptibility. Moreover, mutational processes are influenced by life history and age in both somatic and germline cells.
Now, a new review published in Trends in Genetics discusses recent advances in the evolution of aging at population, organismal and cellular scales.
Biomedical engineers at Duke University have demonstrated that one of the most dangerous mutations found in skin cancers might moonlight as a pathway to mending a broken heart.
The genetic mutation in the protein BRAF, a part of the MAPK signaling pathway that can promote cell division, is one of the most common and most aggressive found in melanoma patients. In a new study, researchers show that introducing this mutation to rat heart tissue grown in a laboratory can induce growth.
Repairing cardiac muscle after a heart attack is the “holy grail” of heart research, complicated by the fact that heart tissue does not regenerate on its own. One potential strategy would be to persuade heart muscle cells to divide by safely delivering a therapeutic gene to patients and fully controlling its activity in the heart.
According to a leak, Arrow Lake-S processors will not increase core count but will require a new chipset.
