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Metformin hijacks AMPK-ERK1/2 signaling to trigger a pathogenic “selection trap” and thymic atrophy

Qin et al. reveal that the diabetes drug metformin sets a “selection trap” in the thymus. It pushes developing immune cells toward maturation while simultaneously triggering their apoptosis via a metabolic stress-induced pathway, challenging the drug’s safety in non-diabetic contexts.

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