Lifeboat Foundation

The universe is still expanding at an accelerating rate, but it may have slowed down recently compared with a few billion years ago, early results from the most precise measurement of its evolution yet suggested Thursday.

The preliminary findings are far from confirmed, but if they hold up, it would further deepen the mystery of dark energy — and likely mean there is something important missing in our understanding of the cosmos.

These signals of our universe’s changing speeds were spotted by the Dark Energy Spectroscopic Instrument, or DESI, which is perched atop a telescope at the Kitt Peak National Observatory in the U.S. state of Arizona.

Using only DNA and glass, researchers made a material four times stronger and five times lighter than steel. It was inspired by Iron Man.

Your immune system should ideally recognize and attack infectious invaders and cancerous cells. But the system requires safety mechanisms, or brakes, to keep it from damaging healthy cells. To do this, T cells—the immune system’s most powerful attackers—rely on immune “checkpoints” to turn immune activation down when they receive the right signal. While these interactions have been well studied, a research team supported in part by NIH has made an unexpected discovery into how a key immune checkpoint works, with potentially important implications for therapies designed to boost or dampen immune activity to treat cancer and autoimmune diseases.¹

The checkpoint in question is a protein called programmed cell death-1 (PD-1). Here’s how it works: PD-1 is a receptor on the surface of T cells, where it latches onto certain proteins, known as PD-L1 and PD-L2, on the surface of other cells in the body. When this interaction occurs, a signal is sent to the T cells that stops them from attacking these other cells.

Cancer cells often take advantage of this braking system, producing copious amounts of PD-L1 on their surface, allowing them to hide from T cells. An effective class of immunotherapy drugs used to treat many cancers works by blocking the interaction between PD-1 and PD-L1, to effectively release the brakes on the immune system to allow the T cells to unleash an assault on cancer cells. Researchers have also developed potential treatments for autoimmune diseases that take the opposite tact: stimulating PD-1 interaction to keep T cells inactive. These PD-1 “agonists” have shown promise in clinical trials as treatments for certain autoimmune diseases.

Researchers from Nottingham Trent University (NTU) have developed realistic 3D printed heart and lung models that can bleed, beat and breathe like their real counterparts.

Designed for organ transplant training, the lifelike models reportedly reflect the tactile qualities of a human heart and can be produced with various tissue hardness levels. Using the models, medical professionals can plan surgeries and safely research and teach transplant procedures, without the risk of complications.

The project, which was led by research fellow Richard Arm, leveraged 3D scans of both healthy and diseased human hearts to 3D print the models to a high level of accuracy.

If you want an electric car that begins charging the moment you plug it into a public fast charger, then you want one with Plug & Charge. Here are the EVs with this feature.

Join us on Patreon! https://www.patreon.com/MichaelLustgartenPhDDiscount Links: Epigenetic, Telomere Testing: https://trudiagnostic.com/?irclickid=U-s3Ii2r7x…

The blackout will give scientists a rare chance to do experiments they cannot do any other time.

A solar occurs when the Moon passes between the Sun and the Earth and blocks the bright light of the Sun’s surface from view. The shadow of the Moon will fall in a small path on Earth’s surface, called the zone of totality.

Credit: SpaceXWatch live as a SpaceX Falcon 9 rocket launches a batch of 21 Starlink internet satellites from Space Launch Complex 4 East at Vandenberg Space…