Attackers are abusing Google Ads and legitimate Claude.ai shared chats in an active malvertising campaign.
Users searching for “Claude mac download” may come across sponsored search results that list claude.ai as the target website, but lead to instructions that install malware on their Mac.
The campaign was spotted by Berk Albayrak, a security engineer at Trendyol Group, who shared his findings on LinkedIn.
Li et al. report that Arabidopsis BSK1 negatively regulates ABA signaling. The stability of BSK1 is dynamically controlled by PUB25 and PUB26, a process likely regulated by BAK1 phosphorylation, whereas UBP24 stabilizes BSK1 by removing ubiquitin chains. ABA shifts this balance toward degradation by inducing the accumulation of PUB25/26.
From a cellular perspective, senescence has been considered a binary state, wherein cells are either senescent or not. This reductionist notion, often defined as irreversible growth arrest, has guided efforts to identify universal biomarkers and senolytics, but both have consistently eluded us. This outcome is not surprising, given that the biological nature of senescence may not be strictly irreversible; the accumulated evidence suggests that growth arrest can become unstable over time, with cells acquiring alterations, occasionally regaining proliferative capacity, or undergoing partial reprogramming, and exhibiting a heterogeneous spectrum of phenotypes (“senotypes”) influenced by tissue types, stressors, temporal dynamics, and disease states.
Our memories of past events are typically not isolated, but they are linked to other related memories. This ability to establish connections between related memories is highly advantageous, as it helps us to recognize familiar patterns in new situations and make predictions that can inform our decisions.
Researchers at UCLA’s Brain Research Institute recently carried out a study on mice aimed at better understanding how the brain decides what memories are connected and which ones are not. Their paper, published in Nature Neuroscience, pinpoints brain regions that could play a role in the organization of memories into coherent pools of knowledge.
“Our lab has long been interested in understanding how the brain connects related memories,” André F. de Sousa, first author of the paper, told Medical Xpress. “In everyday life, new experiences are rarely processed in isolation. Instead, they are often shaped by what we have learned before. This ability allows us to link related events, build knowledge, and use past experiences to guide future behavior. However, this process needs to be carefully controlled.”
A novel, noninvasive brain stimulation approach—known as transcranial temporal interference stimulation (TIs)—may offer a new way to treat motor symptoms in Parkinson’s disease without the need for surgery, according to a pilot study appearing in eBioMedicine. The technique, which uses overlapping electrical currents to selectively target deep brain regions, significantly improved movement in patients compared with a sham treatment when targeting the subthalamic nucleus.
Parkinson’s disease is a progressive neurological disorder that affects movement, often causing tremor, stiffness, and slowed motion. One of the most effective treatments for advanced symptoms is deep brain stimulation (DBS), which involves implanting electrodes into the brain. TIs may be able to achieve a similar effect—targeting the same deep brain structures —but entirely from outside the skull, using carefully calibrated electrical fields delivered through the scalp.
In the randomized, double-blind, crossover study, titled “Transcranial temporal interference stimulation targeting the subthalamic region for motor symptoms in Parkinson’s disease: a pilot, randomised, double-blind, sham-controlled crossover study,” 30 people with early-to mid-stage Parkinson’s disease received a single 20-minute session of individualized TIs targeting the subthalamic region—a key node in the brain’s motor control network—as well as a sham or placebo treatment in a separate session.
For many people who smoke, quitting is not just a matter of willpower. It is a tug-of-war in the brain—between the pull of reward and the ability to resist.
A study published in the Journal of Psychiatric Research suggests that shifting that balance may be possible. Using a noninvasive brain stimulation technique called repetitive transcranial magnetic stimulation, or rTMS, researchers at MUSC Hollings Cancer Center found that stimulating a specific brain region that regulates self-control significantly reduced how much people smoked.
When danger lurks, instinct keeps us safe. It compels us to run from a burning building or wrestle a knife-wielding attacker to the ground. It also adjusts our body physiology to support these behaviors.
Survival helps explain why. But the mechanisms that link the brain and the body—the “switch” between rest and action—have long been shrouded in mystery.
A research team at Rutgers University-New Brunswick thinks they may have identified a key mechanism, and the findings may hold important clues to how diverse neurological conditions, such as alcohol use disorder and Parkinson’s, could be diagnosed and treated.